Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 301
... occur in patients with inflammatory bowel disease ( IBD ) or the irritable bowel syndrome ( IBS ) .1 Diarrhea is the most common symptom in IBD , occurring in approxi- mately 90 % of patients with Crohn's disease ( CD ) 2 or ulcerative ...
... occur in patients with inflammatory bowel disease ( IBD ) or the irritable bowel syndrome ( IBS ) .1 Diarrhea is the most common symptom in IBD , occurring in approxi- mately 90 % of patients with Crohn's disease ( CD ) 2 or ulcerative ...
Page 509
... occur in toxic megacolon . These ulcers may undermine the adjacent mucosa and can occasionally be branching . They have a superficial layer of necrotic debris and slough , with underlying inflamed granulation tissue . The key to ...
... occur in toxic megacolon . These ulcers may undermine the adjacent mucosa and can occasionally be branching . They have a superficial layer of necrotic debris and slough , with underlying inflamed granulation tissue . The key to ...
Page 510
... occur in UC . The exceptions to this statement are instances when previous subtotal perforation has taken place and cases of UC associated with diverticular disease . Backwash Ileitis Backwash ileitis can occur in up to 20 % of patients ...
... occur in UC . The exceptions to this statement are instances when previous subtotal perforation has taken place and cases of UC associated with diverticular disease . Backwash Ileitis Backwash ileitis can occur in up to 20 % of patients ...
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Common terms and phrases
abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro