Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 61
... organisms into the Peyer's patch ; however , organisms that typically enter through M cells ( po- liovirus , reovirus , Giardia ) do not usually result in aphthous ulcer formation . Therefore , either the nature of the organism or the ...
... organisms into the Peyer's patch ; however , organisms that typically enter through M cells ( po- liovirus , reovirus , Giardia ) do not usually result in aphthous ulcer formation . Therefore , either the nature of the organism or the ...
Page 265
... organism . 15 The very small amounts of DNA obtained from most of these organisms , however , preclude even conventional DNA hybridization as a method of identification . DNA Hybridization Studies The extremely long incubation periods ...
... organism . 15 The very small amounts of DNA obtained from most of these organisms , however , preclude even conventional DNA hybridization as a method of identification . DNA Hybridization Studies The extremely long incubation periods ...
Page 269
... organisms present in the disease may be responsible for resistance to treatment . Treatment of M. paratuberculosis infection in pri- mates with ileitis was successful in eliminating the organism and curing the disease.20 It is hoped ...
... organisms present in the disease may be responsible for resistance to treatment . Treatment of M. paratuberculosis infection in pri- mates with ileitis was successful in eliminating the organism and curing the disease.20 It is hoped ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro