Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 206
... plasma determinations of eicosanoids are often artifactual , because synthesis may occur during blood - letting . Consequently , plasma concentrations may vary by orders of magnitude.3 In vivo formation of eicosanoids has therefore been ...
... plasma determinations of eicosanoids are often artifactual , because synthesis may occur during blood - letting . Consequently , plasma concentrations may vary by orders of magnitude.3 In vivo formation of eicosanoids has therefore been ...
Page 541
... plasma and body tissues.103 As glutamine can be synthesized in many tissues , it is considered a nonessential amino acid . The first evidence that the intestine metabolized glutamine appeared in 1960 , when Finch and Hird demonstrated ...
... plasma and body tissues.103 As glutamine can be synthesized in many tissues , it is considered a nonessential amino acid . The first evidence that the intestine metabolized glutamine appeared in 1960 , when Finch and Hird demonstrated ...
Page 551
... plasma glutamine by the small intestine . J Biol Chem 1974 ; 249 : 5070-5079 . 102. Settle GR . Short - chain fatty acids and their potential role in nutritional support . J Parenter Enteral Nutr 1988 ; 12 ( suppl . ) : 104S - 107S ...
... plasma glutamine by the small intestine . J Biol Chem 1974 ; 249 : 5070-5079 . 102. Settle GR . Short - chain fatty acids and their potential role in nutritional support . J Parenter Enteral Nutr 1988 ; 12 ( suppl . ) : 104S - 107S ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro