Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 317
... present in inflammatory bowel disease on mucin secretion are largely un- known . Although histamine may elicit mucin release , prostaglandins are not known to have effects on mucin secretion in the intestines . Despite their effects on ...
... present in inflammatory bowel disease on mucin secretion are largely un- known . Although histamine may elicit mucin release , prostaglandins are not known to have effects on mucin secretion in the intestines . Despite their effects on ...
Page 511
... present in the deepest part of the lamina propria . After 4 days , some cases of acute self - limited colitis developed these features to a lesser degree so that they became nondiscriminatory . Lymphocytic ( Microscopic ) Colitis ...
... present in the deepest part of the lamina propria . After 4 days , some cases of acute self - limited colitis developed these features to a lesser degree so that they became nondiscriminatory . Lymphocytic ( Microscopic ) Colitis ...
Page 514
... present . Microscopy shows ulceration , transmural chronic inflammation with or without submucosal lymphoid aggregates , and fissurelike ulcers . Obviously , this lesion can occasionally be quite difficult to distinguish from CD ...
... present . Microscopy shows ulceration , transmural chronic inflammation with or without submucosal lymphoid aggregates , and fissurelike ulcers . Obviously , this lesion can occasionally be quite difficult to distinguish from CD ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro