Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
From inside the book
Results 1-3 of 83
Page 28
... results less powerful ) . Again , these associa- tions are inconsistent . Fewer studies have examined the HLA - DR locus , with a range of results ; the clearest result has been reported in the Japanese population , with a dramatic ...
... results less powerful ) . Again , these associa- tions are inconsistent . Fewer studies have examined the HLA - DR locus , with a range of results ; the clearest result has been reported in the Japanese population , with a dramatic ...
Page 62
... result in increased T - cell activation ( predominantly CD4 + T cells ) and an immunologic cascade of chronic inflammation . As the mucosal barrier is altered in IBD , epithelial cell access to a larger number of luminal antigens may result ...
... result in increased T - cell activation ( predominantly CD4 + T cells ) and an immunologic cascade of chronic inflammation . As the mucosal barrier is altered in IBD , epithelial cell access to a larger number of luminal antigens may result ...
Page 280
... result of depletion of the bile salt pool , the decreased concentration of bile salts in the proximal small intestine causes deficient micellar lipid . The result is increased passage of fatty acids into the colon and the production of ...
... result of depletion of the bile salt pool , the decreased concentration of bile salts in the proximal small intestine causes deficient micellar lipid . The result is increased passage of fatty acids into the colon and the production of ...
Other editions - View all
Common terms and phrases
abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro