Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 7
... shown any significant difference from other ethnic groups . 59 Therefore , a special suscep- tibility for inflammatory bowel disease among Jews has not been established . Evidence from well - designed and sufficiently large studies is ...
... shown any significant difference from other ethnic groups . 59 Therefore , a special suscep- tibility for inflammatory bowel disease among Jews has not been established . Evidence from well - designed and sufficiently large studies is ...
Page 288
... shown to stimulate sodium and chloride absorption 191,192 and inhibit HCO3 secretion192 in rabbit ileum in vitro and to inhibit fluid secretion in rat jejunum in vivo , possibly by blocking the stimulation of adenylate cyclase . 193 ...
... shown to stimulate sodium and chloride absorption 191,192 and inhibit HCO3 secretion192 in rabbit ileum in vitro and to inhibit fluid secretion in rat jejunum in vivo , possibly by blocking the stimulation of adenylate cyclase . 193 ...
Page 369
... shown to reflect clinical disease activity . They are widely used in Europe , but are un- available in most centers in the United States.25,26 Intestinal inflammation may result in an enteric loss of proteins . Quantitative fecal loss ...
... shown to reflect clinical disease activity . They are widely used in Europe , but are un- available in most centers in the United States.25,26 Intestinal inflammation may result in an enteric loss of proteins . Quantitative fecal loss ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro