Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 210
... stimulation of the cells with the calcium ionophore A - 23187.8 The findings of markedly enhanced in vivo LTB4 formation in UC36 together with its known biologic effects ( especially its chemotactic and chemokinetic properties ) suggest ...
... stimulation of the cells with the calcium ionophore A - 23187.8 The findings of markedly enhanced in vivo LTB4 formation in UC36 together with its known biologic effects ( especially its chemotactic and chemokinetic properties ) suggest ...
Page 277
... stimulating intestinal epithelial secretion . These cells , which directly underlie the basal aspect of epithelial cells , can produce AA metabolites on stimulation with agents such as bradykinin and calcium ionophore ( M. J. Ciancio ...
... stimulating intestinal epithelial secretion . These cells , which directly underlie the basal aspect of epithelial cells , can produce AA metabolites on stimulation with agents such as bradykinin and calcium ionophore ( M. J. Ciancio ...
Page 305
... stimulation . Indomethacin ( 5 mg / kg IV ) , administered 2 hours before the myoelectric recordings , did not increase baseline and bethanechol - stimulated colonic spike activity in the animals with colitis . Other studies show that ...
... stimulation . Indomethacin ( 5 mg / kg IV ) , administered 2 hours before the myoelectric recordings , did not increase baseline and bethanechol - stimulated colonic spike activity in the animals with colitis . Other studies show that ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro