Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 222
... sulfasalazine . Biochem Pharmacol 1983 ; 32 : 2205-2209 . 111. Hawkey CJ , Boughton - Smith NK , Whittle BJR . Modulation of human colonic arach- idonic acid metabolism by sulfasalazine . Dig Dis Sci 1985 ; 30 : 1161-1165 . 112. Stenson ...
... sulfasalazine . Biochem Pharmacol 1983 ; 32 : 2205-2209 . 111. Hawkey CJ , Boughton - Smith NK , Whittle BJR . Modulation of human colonic arach- idonic acid metabolism by sulfasalazine . Dig Dis Sci 1985 ; 30 : 1161-1165 . 112. Stenson ...
Page 420
... Sulfasalazine Although there are data to suggest that sulfasalazine is most effective in mild ulcerative colitis or Crohn's colitis , most gastroenterologists still tend to use sulfasalazine ( Azulfidine ) in the therapy of mild to ...
... Sulfasalazine Although there are data to suggest that sulfasalazine is most effective in mild ulcerative colitis or Crohn's colitis , most gastroenterologists still tend to use sulfasalazine ( Azulfidine ) in the therapy of mild to ...
Page 421
... sulfasalazine . Renal tubular function also appears unaffected 1 year after initiation of sulfasalazine.9 Sulfasalazine does not appear to have a tera- togenic effect on the fetus although its metabolites readily cross the placental ...
... sulfasalazine . Renal tubular function also appears unaffected 1 year after initiation of sulfasalazine.9 Sulfasalazine does not appear to have a tera- togenic effect on the fetus although its metabolites readily cross the placental ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro