Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 599
Richard P. MacDermott, William F. Stenson. Chapter 30 Surgical Management of Ulcerative Colitis James M. Becker , MD Greater than 50 % of patients with chronic ulcerative colitis come to surgery within the first 10 years of their illness ...
Richard P. MacDermott, William F. Stenson. Chapter 30 Surgical Management of Ulcerative Colitis James M. Becker , MD Greater than 50 % of patients with chronic ulcerative colitis come to surgery within the first 10 years of their illness ...
Page 620
... surgery can be per- formed.18 The need for surgery may not have been eliminated ( older studies claim that TPN did not change the natural history of the disease ) , 19 but the nature of the requisite surgical procedures has been changed ...
... surgery can be per- formed.18 The need for surgery may not have been eliminated ( older studies claim that TPN did not change the natural history of the disease ) , 19 but the nature of the requisite surgical procedures has been changed ...
Page 627
... surgery should nevertheless be done , as there is a greater than 90 % probability that resection will eventually be necessary . It is too hazardous to ... Surgery : Colonic Crohn's Disease Internal SURGICAL MANAGEMENT OF CROHN'S DISEASE 627.
... surgery should nevertheless be done , as there is a greater than 90 % probability that resection will eventually be necessary . It is too hazardous to ... Surgery : Colonic Crohn's Disease Internal SURGICAL MANAGEMENT OF CROHN'S DISEASE 627.
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro