Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 455
... terminal ileum , glucagon is administered intravenously , and air is insufflated into the colon and refluxed into the terminal ileum ( Fig . 25.2 ) . A successful double - contrast examination of the terminal ileum is obtained in ...
... terminal ileum , glucagon is administered intravenously , and air is insufflated into the colon and refluxed into the terminal ileum ( Fig . 25.2 ) . A successful double - contrast examination of the terminal ileum is obtained in ...
Page 461
... terminal ileum in approximately 55 % of patients , the terminal ileum alone in 14 % , the terminal ileum extending proximally in 13 % , the colon alone in 15 % , and the proximal small bowel with terminal ileum sparing in 3 % .20-23 ...
... terminal ileum in approximately 55 % of patients , the terminal ileum alone in 14 % , the terminal ileum extending proximally in 13 % , the colon alone in 15 % , and the proximal small bowel with terminal ileum sparing in 3 % .20-23 ...
Page 474
... terminal ileum with extrinsic involvement of the sigmoid colon leading to an ileosigmoid fistula . ( A ) Barium - filled tracks ( arrowheads ) extend from the narrowed and cobblestoned terminal ileum ( T ) to the sigmoid colon ( S ) ...
... terminal ileum with extrinsic involvement of the sigmoid colon leading to an ileosigmoid fistula . ( A ) Barium - filled tracks ( arrowheads ) extend from the narrowed and cobblestoned terminal ileum ( T ) to the sigmoid colon ( S ) ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro