Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 118
... tested in vitro.201 Moreover , an increased number of mononuclear cells producing IFN - y were recently found in CD lesions compared with normal mucosa , 202 although others have not seen this in either CD or UC.2 203,204 An increased ...
... tested in vitro.201 Moreover , an increased number of mononuclear cells producing IFN - y were recently found in CD lesions compared with normal mucosa , 202 although others have not seen this in either CD or UC.2 203,204 An increased ...
Page 368
... tests lies in their lack of sub- jectivity ; however , their disadvantage relates to their lack of specificity . In gen- eral , there are three types of tests for evaluating inflammation : serologic acute- phase reactants , measures of ...
... tests lies in their lack of sub- jectivity ; however , their disadvantage relates to their lack of specificity . In gen- eral , there are three types of tests for evaluating inflammation : serologic acute- phase reactants , measures of ...
Page 413
... tests in a patient with concomitant inflammatory bowel disease . We have out- lined a scheme in Figure 21.1 . For patients with predominantly hepatocellular disease initial screening should include serology for autoantibodies , serum im ...
... tests in a patient with concomitant inflammatory bowel disease . We have out- lined a scheme in Figure 21.1 . For patients with predominantly hepatocellular disease initial screening should include serology for autoantibodies , serum im ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro