Inflammatory Bowel DiseaseRichard P. MacDermott, William F. Stenson |
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Page 90
... vitro studies of ulcerative colitis . I. Reactions of pa- tients ' serum with human fetal colon cells in tissue cultures . J Exp Med 1963 ; 117 : 705-715 . 51. Rabin BS , Rogers SJ . Nonpathogenicity of anti - intestinal antibody in the ...
... vitro studies of ulcerative colitis . I. Reactions of pa- tients ' serum with human fetal colon cells in tissue cultures . J Exp Med 1963 ; 117 : 705-715 . 51. Rabin BS , Rogers SJ . Nonpathogenicity of anti - intestinal antibody in the ...
Page 199
... vitro by an indirect rosetting technique . J Immunol 1986 ; 136 : 4515-4524 . 52. Butterfield JH , Weider DA . In vitro sensitivity of immature mast cells to chemother- apeutic agents . Int Arch Allergy Appl Immunol 1989 ; 89 : 297–300 ...
... vitro by an indirect rosetting technique . J Immunol 1986 ; 136 : 4515-4524 . 52. Butterfield JH , Weider DA . In vitro sensitivity of immature mast cells to chemother- apeutic agents . Int Arch Allergy Appl Immunol 1989 ; 89 : 297–300 ...
Page 213
... vitro observations of increased PGE2 and PGI2 production by normal colonic mucosa in the presence of 5 - ASA , in addition to decreased enzymatic degradation of PGs in the presence of sulfasalazine ( but not 5 - ASA ) . 73,106-109 ...
... vitro observations of increased PGE2 and PGI2 production by normal colonic mucosa in the presence of 5 - ASA , in addition to decreased enzymatic degradation of PGs in the presence of sulfasalazine ( but not 5 - ASA ) . 73,106-109 ...
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abnormalities acid action activity addition agents alterations antibodies antigens appear associated bacterial blood cause chronic Clin clinical colonic compared complement complex component Crohn's disease cytokines cytotoxic decreased defect determined diarrhea effects enhanced epithelial cells epithelium et al evidence expression factor findings function Gastroenterology genes genetic granulomas growth human immune immune response Immunol immunologic important incidence increased induced infection inflammation inflammatory bowel disease inhibition initially injury intestinal involved isolated lamina propria lesions leukocyte levels lymphocytes macrophages markers mast cells mechanisms mediators mononuclear mucin mucosal Nature neutrophils normal observed occur patients population possible potential present production prostaglandin protein rates recent receptor regulation relatives release reported response risk role schistosomiasis secretion secretory serum shown specific stimulation studies suggest T-cell tissue ulcerative colitis vitro