The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3 |
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Page 26
The inferior surface , below the attachment of the transverse mesocolon , is
related to the duodenojejunal junction and to the splenic flexure of the colon .
The posterior surface is in contact with the aorta , the splenic vein and the left
kidney ...
The inferior surface , below the attachment of the transverse mesocolon , is
related to the duodenojejunal junction and to the splenic flexure of the colon .
The posterior surface is in contact with the aorta , the splenic vein and the left
kidney ...
Page 105
They hatch when they fall into fresh water . The larvae or miracidia survive only a
few hours , unless they can attach themselves to snails , which they penetrate . In
the snail's digestive gland the larvae pass through several stages ( sporocysts ) ...
They hatch when they fall into fresh water . The larvae or miracidia survive only a
few hours , unless they can attach themselves to snails , which they penetrate . In
the snail's digestive gland the larvae pass through several stages ( sporocysts ) ...
Page 130
In other instances edema of the wall is supposed to block the blood supply , and
in exceptional cases torsion of the gallbladder has been found as a result of an
abnormal peritoneal attachment to the liver ( see page 123 ) . Not infrequently ...
In other instances edema of the wall is supposed to block the blood supply , and
in exceptional cases torsion of the gallbladder has been found as a result of an
abnormal peritoneal attachment to the liver ( see page 123 ) . Not infrequently ...
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Contents
PHYSIOLOGY AND PATHOPHYSIOLOGY OF | 1 |
Cholelithiasis I Stone Formation | 2 |
Cholecystitis II Complications | 8 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear AREA associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause celiac cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenal duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty fibers flow formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice later leads less liver cells lobe lobular lymphatic necrosis nerves nodes nodules normal obstruction occur organ origin pain pancreatic duct passes patients pigment plates portal vein portion posterior present produce protein rare result serum severe sinusoids sometimes space sphincter splenic stage stones structures SUPERIOR MESENTERIC surface tests tion tissue tract tumor urine usually vary vessels wall