The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3 |
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Page 62
These cell remnants are then expelled from the liver plate and lie in the tissue
spaces as acidophilic masses , the so - called " Councilman bodies " , named
because of the fact that similar formations were discovered by Councilman in
yellow ...
These cell remnants are then expelled from the liver plate and lie in the tissue
spaces as acidophilic masses , the so - called " Councilman bodies " , named
because of the fact that similar formations were discovered by Councilman in
yellow ...
Page 88
... the ferric iron is bound to a protein complex , apoferritin , which apparently is
synthesized by mucosal epithelium . The iron - protein complex - now called
ferritin – is the main organic storage form in the body , but before being taken up
by the ...
... the ferric iron is bound to a protein complex , apoferritin , which apparently is
synthesized by mucosal epithelium . The iron - protein complex - now called
ferritin – is the main organic storage form in the body , but before being taken up
by the ...
Page 116
The so - called hepatorenal syndrome ( see page 76 ) , a frequent complication ,
has been associated with a toxic effect from tissue breakdown products from the
liver . However , traumatized , necrotic and even completely separated hepatic ...
The so - called hepatorenal syndrome ( see page 76 ) , a frequent complication ,
has been associated with a toxic effect from tissue breakdown products from the
liver . However , traumatized , necrotic and even completely separated hepatic ...
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Contents
PHYSIOLOGY AND PATHOPHYSIOLOGY OF | 1 |
Cholelithiasis I Stone Formation | 2 |
Cholecystitis II Complications | 8 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear AREA associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause celiac cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenal duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty fibers flow formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice later leads less liver cells lobe lobular lymphatic necrosis nerves nodes nodules normal obstruction occur organ origin pain pancreatic duct passes patients pigment plates portal vein portion posterior present produce protein rare result serum severe sinusoids sometimes space sphincter splenic stage stones structures SUPERIOR MESENTERIC surface tests tion tissue tract tumor urine usually vary vessels wall