The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
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Page 25
... develop in both buds but anastomose when the buds interlock . The secretion of neck , body and tail is subsequently shunted into the duct of the smaller ventral pancreas , which thus becomes the principal pancreatic duct of Wirsung ...
... develop in both buds but anastomose when the buds interlock . The secretion of neck , body and tail is subsequently shunted into the duct of the smaller ventral pancreas , which thus becomes the principal pancreatic duct of Wirsung ...
Page 66
... DEVELOP BETWEEN AREAS OF IRREGULARLY DISTRIBUTED FAT . MICROMEMBRANES DEVELOP IN THESE " STRESS FISSURES " ( A ) AS WELL AS AROUND FATTY CYSTS ( B ) AND NECROTIC AREAS ( C ) AND RADIATING FROM PORTAL TRIADS ( D ) . REGENERATION STARTS ...
... DEVELOP BETWEEN AREAS OF IRREGULARLY DISTRIBUTED FAT . MICROMEMBRANES DEVELOP IN THESE " STRESS FISSURES " ( A ) AS WELL AS AROUND FATTY CYSTS ( B ) AND NECROTIC AREAS ( C ) AND RADIATING FROM PORTAL TRIADS ( D ) . REGENERATION STARTS ...
Page 137
... develop , but , even more frequently , fibrosis replaces within several months the acinar tis- sue , leaving , as a rule , the islands of Langerhans intact , so that diabetes develops late , if at all . Metas- SLOUGHING TUMOR MASSES ...
... develop , but , even more frequently , fibrosis replaces within several months the acinar tis- sue , leaving , as a rule , the islands of Langerhans intact , so that diabetes develops late , if at all . Metas- SLOUGHING TUMOR MASSES ...
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Common terms and phrases
abdominal abnormal abscesses acid acute pancreatitis Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatic MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organs pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic SECTION septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tumor urine urobilinogen usually vascular vena cava vessels viral hepatitis wall