The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
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Page 40
... factors necessary for the conversion of prothrombin into thrombin ( labile factor V , also called proaccelerin or AC globulin , and stable factor VII , also called convertin or cothromboplastin ) . Deficiency effects of these factors in ...
... factors necessary for the conversion of prothrombin into thrombin ( labile factor V , also called proaccelerin or AC globulin , and stable factor VII , also called convertin or cothromboplastin ) . Deficiency effects of these factors in ...
Page 77
... FACTORS E 3 . MICROCALCULI IN DILATED BILE DUCTULES 1 . CENTRILOBULAR. NUTRITIONAL LIVER DISEASES ALCOHOL SUGAR INCREASED METABOLIC NEEDS FAT DECREASED METABOLIC NEEDS PROTEIN LIPOTROPIC FACTORS Starvation , Malnutrition Hepatic injury ...
... FACTORS E 3 . MICROCALCULI IN DILATED BILE DUCTULES 1 . CENTRILOBULAR. NUTRITIONAL LIVER DISEASES ALCOHOL SUGAR INCREASED METABOLIC NEEDS FAT DECREASED METABOLIC NEEDS PROTEIN LIPOTROPIC FACTORS Starvation , Malnutrition Hepatic injury ...
Page 79
... factors , e.g. , acute and chronic episodes of hepatic necrosis , are responsible for the transition of the fatty human liver into cirrhosis ( see page 66 ) . In the temperate zone such epi- sodes may be provoked by infections elsewhere ...
... factors , e.g. , acute and chronic episodes of hepatic necrosis , are responsible for the transition of the fatty human liver into cirrhosis ( see page 66 ) . In the temperate zone such epi- sodes may be provoked by infections elsewhere ...
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Common terms and phrases
abdominal abnormal abscesses acid acute pancreatitis Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatic MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organs pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic SECTION septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tumor urine urobilinogen usually vascular vena cava vessels viral hepatitis wall