The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
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Page 2
... later in one - cell - thick plates . The endo- thelial cells of the plexus become the Kupffer cells . The mesenchyma provides also the connective tissue for the capsule and the portal tracts . At 10 - mm . embryo- nal length , bile ...
... later in one - cell - thick plates . The endo- thelial cells of the plexus become the Kupffer cells . The mesenchyma provides also the connective tissue for the capsule and the portal tracts . At 10 - mm . embryo- nal length , bile ...
Page 116
... Later , the effects of biliary peritonitis , following laceration of bile ducts or shock or infections , become important causes of death . The so - called hepatorenal syndrome ( see page 76 ) , a frequent complication , has been ...
... Later , the effects of biliary peritonitis , following laceration of bile ducts or shock or infections , become important causes of death . The so - called hepatorenal syndrome ( see page 76 ) , a frequent complication , has been ...
Page 144
... later stages the pain becomes more and more persistent . Ingestion of any food may provoke an attack with nausea and vomiting , so that it is not surprising that patients with relapsing pancreatitis become seriously malnourished . In ...
... later stages the pain becomes more and more persistent . Ingestion of any food may provoke an attack with nausea and vomiting , so that it is not surprising that patients with relapsing pancreatitis become seriously malnourished . In ...
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Common terms and phrases
abdominal abnormal abscesses acid acute pancreatitis Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatic MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organs pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic SECTION septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tumor urine urobilinogen usually vascular vena cava vessels viral hepatitis wall