The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
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Page 64
... NECROSIS Necrosis implies not only death of cells but also the phenomena following cell death , namely , the disappearance of cells and frequently also the environmental reactions to and the vanishing of dead cells . The final and ...
... NECROSIS Necrosis implies not only death of cells but also the phenomena following cell death , namely , the disappearance of cells and frequently also the environmental reactions to and the vanishing of dead cells . The final and ...
Page 65
... NECROSIS MASSIVE NECROSIS CENTRAL NECROSIS SUBMASSIVE NECROSIS B SUBMASSIVE COLLAPSE PERIPORTAL ( PERIPHERAL ) ONECROSIS O CENTRAL NECROSIS WITH BRIDGING B SUBMASSIVE NECROSIS #Natter M.D ®CIBA e morpho nd the inci ently hazy V may be ...
... NECROSIS MASSIVE NECROSIS CENTRAL NECROSIS SUBMASSIVE NECROSIS B SUBMASSIVE COLLAPSE PERIPORTAL ( PERIPHERAL ) ONECROSIS O CENTRAL NECROSIS WITH BRIDGING B SUBMASSIVE NECROSIS #Natter M.D ®CIBA e morpho nd the inci ently hazy V may be ...
Page 143
... necrosis . Diabetes may become evident during the acute phase , and permanent if patients with necrosis of the gland survive . Edema occluding the common bile duct or , in later phases , scar tissue may cause jaun- dice . Splenic vein ...
... necrosis . Diabetes may become evident during the acute phase , and permanent if patients with necrosis of the gland survive . Edema occluding the common bile duct or , in later phases , scar tissue may cause jaun- dice . Splenic vein ...
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abdominal abnormal abscesses acid acute pancreatitis Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatic MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organs pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic SECTION septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tumor urine urobilinogen usually vascular vena cava vessels viral hepatitis wall