The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3 |
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Page 85
Experimentally , amyloidosis has been produced by feeding large amounts of
protein , especially cheese or casein , and it has been claimed that the disease is
rare in countries with little milk or cheese consumption , whereas it is a frequent ...
Experimentally , amyloidosis has been produced by feeding large amounts of
protein , especially cheese or casein , and it has been claimed that the disease is
rare in countries with little milk or cheese consumption , whereas it is a frequent ...
Page 86
Galactosemia , another rare , inborn disturbance of the carbohydrate metabolism
, is assumedly caused by a defect of a hepatic enzyme required for the
transformation of galactose - 1 - phosphate to glucose - 1 - phosphate . Excessive
...
Galactosemia , another rare , inborn disturbance of the carbohydrate metabolism
, is assumedly caused by a defect of a hepatic enzyme required for the
transformation of galactose - 1 - phosphate to glucose - 1 - phosphate . Excessive
...
Page 141
It is rare that more than a single nodule can be detected . The structure is usually
similar to that of the normal pancreas but may become cystic , and instances
where the mass was primarily or entirely made up of islet cells have been
reported .
It is rare that more than a single nodule can be detected . The structure is usually
similar to that of the normal pancreas but may become cystic , and instances
where the mass was primarily or entirely made up of islet cells have been
reported .
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Contents
PHYSIOLOGY AND PATHOPHYSIOLOGY OF | 1 |
Cholelithiasis I Stone Formation | 2 |
Cholecystitis II Complications | 8 |
Copyright | |
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear AREA associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause celiac cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued course cystic duct cysts cytoplasm damage develop diagnosis disease duodenal duodenum elevated enlarged enter especially excretion extends extrahepatic factors fatty fibers flow formation frequently function gallbladder gland glucose head hepatic artery hepatic duct increased infection inferior injury instances intestinal involved jaundice later leads less liver cells lobe lobular lymphatic necrosis nerves nodes nodules normal obstruction occur organ origin pain pancreatic duct passes patients pigment plates portal vein portion posterior present produce protein rare result serum severe sinusoids sometimes space sphincter splenic stage stones structures SUPERIOR MESENTERIC surface tests tion tissue tract tumor urine usually vary vessels wall