The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
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Page 68
... become anastomoses . The liver cells in those parts of the lobules that have been separated from their original parenchymal environment also start to multiply , and this regenerative effort results in cell plates which rearrange ...
... become anastomoses . The liver cells in those parts of the lobules that have been separated from their original parenchymal environment also start to multiply , and this regenerative effort results in cell plates which rearrange ...
Page 69
... become more and more dilated but are seen only at operation or autopsy . The veins in the anterior abdominal wall dilate as a result of collaterals developing around the remnants of the fetal circula- tion in the round ligament . They ...
... become more and more dilated but are seen only at operation or autopsy . The veins in the anterior abdominal wall dilate as a result of collaterals developing around the remnants of the fetal circula- tion in the round ligament . They ...
Page 111
... become pedunculated . The irregularity of the structural elements ( liver cell plates not arranged around a central vein , prolifer- ated bile ducts and connective tissue ) and the occasional predominance of one or the other component ...
... become pedunculated . The irregularity of the structural elements ( liver cell plates not arranged around a central vein , prolifer- ated bile ducts and connective tissue ) and the occasional predominance of one or the other component ...
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fatty fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall