The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
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Page 56
... CHANGES IN ACUTE PANCREATITIS AMYLASE LIPASE DIGESTION BY TRYPSIN CALCIUM The most characteristic changes in acute pancreatitis ( see page 143 ) and the most useful in diagnosis are the eleva- tions of amylase and lipase in serum or ...
... CHANGES IN ACUTE PANCREATITIS AMYLASE LIPASE DIGESTION BY TRYPSIN CALCIUM The most characteristic changes in acute pancreatitis ( see page 143 ) and the most useful in diagnosis are the eleva- tions of amylase and lipase in serum or ...
Page 87
... changes , but the nitrogen loss is quanti- tatively not great enough to produce significant malnutrition . The hepatic changes in this disease are extremely vari- able . In biopsy specimens only fatty metamorphosis and portal fibrosis ...
... changes , but the nitrogen loss is quanti- tatively not great enough to produce significant malnutrition . The hepatic changes in this disease are extremely vari- able . In biopsy specimens only fatty metamorphosis and portal fibrosis ...
Page 163
... changes , 75 , 86 , 87 , 119 , 120 mercapturic acid , 44 , 64 mesentery , see peritoneum mesobilirubinogen , 47 mesocolon ( see also peritoneum ) , 28 , 29 , 131 , 145 mesoduodenum ( see also peritoneum ) , 25 methionine , 37 methyl ...
... changes , 75 , 86 , 87 , 119 , 120 mercapturic acid , 44 , 64 mesentery , see peritoneum mesobilirubinogen , 47 mesocolon ( see also peritoneum ) , 28 , 29 , 131 , 145 mesoduodenum ( see also peritoneum ) , 25 methionine , 37 methyl ...
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fatty fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall