The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
From inside the book
Results 1-3 of 8
Page 22
... folds and lined by tall columnar surface epithelium ; ( 2 ) a fibromuscular layer ; ( 3 ) a subserous adventitia ; and ( 4 ) a serosal layer , men- tioned above . The irregular folds , easily seen in contracted state , disappear on ...
... folds and lined by tall columnar surface epithelium ; ( 2 ) a fibromuscular layer ; ( 3 ) a subserous adventitia ; and ( 4 ) a serosal layer , men- tioned above . The irregular folds , easily seen in contracted state , disappear on ...
Page 127
... FOLDS , FLATTENING OF EPITHELIUM HYDROPS AND EMPYEMA OF GALLBLADDER A gallstone obstructing the cystic duct usually elicits a biliary colic , which sub- sides sometimes with persistent impac- tion of the stone either in the cystic duct ...
... FOLDS , FLATTENING OF EPITHELIUM HYDROPS AND EMPYEMA OF GALLBLADDER A gallstone obstructing the cystic duct usually elicits a biliary colic , which sub- sides sometimes with persistent impac- tion of the stone either in the cystic duct ...
Page 129
... folds of the gallbladder , which lose their regularity and normal shape . Such lipid - laden folds may become the nucleus for a cholesterol stone , or a great number of foam cells may extend through the entire mucosa beyond the folds ...
... folds of the gallbladder , which lose their regularity and normal shape . Such lipid - laden folds may become the nucleus for a cholesterol stone , or a great number of foam cells may extend through the entire mucosa beyond the folds ...
Other editions - View all
Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fatty fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall