The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3 |
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Page 40
NORMAL PROTHROMBIN FORMATION VITAMIN K IN FOOD MILLON'S
REACTION FOR TYROSINE ♡ 6 . AMINO - ACIDURIA PROTEIN IN FOOD
PAPER CHROMATOGRAM PROTHROMBIN PROTHROMBIN FORMED IN
LIVERCLOT ...
NORMAL PROTHROMBIN FORMATION VITAMIN K IN FOOD MILLON'S
REACTION FOR TYROSINE ♡ 6 . AMINO - ACIDURIA PROTEIN IN FOOD
PAPER CHROMATOGRAM PROTHROMBIN PROTHROMBIN FORMED IN
LIVERCLOT ...
Page 68
The increasing disturbance of the hepatic circulation in the lobular and nodular
parenchyma is also a stimulus for central necrosis as well as peripheral
inflammation , both of which , in turn , provoke septa formation and separation of
single or ...
The increasing disturbance of the hepatic circulation in the lobular and nodular
parenchyma is also a stimulus for central necrosis as well as peripheral
inflammation , both of which , in turn , provoke septa formation and separation of
single or ...
Page 74
PORTAL HYPERTENSION 3 HEART FAILURE ASCITES Pathogenesis 2 LIVER
DAMAGE A - HYPOPROTEINEMIA ( DECREASED ALBUMIN FORMATION ) B -
POSSIBLY DECREASED INACTIVATION OF ANTIDIURETIC HORMONE .
PORTAL HYPERTENSION 3 HEART FAILURE ASCITES Pathogenesis 2 LIVER
DAMAGE A - HYPOPROTEINEMIA ( DECREASED ALBUMIN FORMATION ) B -
POSSIBLY DECREASED INACTIVATION OF ANTIDIURETIC HORMONE .
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Contents
SECTION XV | 1 |
Cellular Elements of Liver | 7 |
Vessel and Duct Distribution | 13 |
Copyright | |
14 other sections not shown
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Common terms and phrases
abnormal abscesses acid activity acute acute pancreatitis alterations appear areas associated become biliary biliary tract bilirubin biopsy blood body branches carcinoma cause cent central changes cholesterol chronic CIBA cirrhosis clinical common bile duct complete connective Continued cystic duct cysts cytoplasm damage degeneration depending develop diagnosis dilated disease duodenum effect elevated enlarged enter especially excretion extends extrahepatic factors failure fatty fibrosis findings formation frequently function gallbladder gland glucose hepatic artery increased indicate infection inferior injury instances intestinal involved jaundice Kupffer cells latter leads lesions less liver cells lobular lobule manifestations necrosis nodes nodules normal observed obstruction occur organs origin pain pancreatic patients period picture pigment PLATE portal vein present pressure primary produce protein rare result seen serum severe sometimes space sphincter stage stones structures superior surface surrounding tests tion tissue tract tumor usually vary vessels viral wall