The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
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Page 7
... nucleus , however , still remaining in the center . Subsequently , the droplets merge , and one large drop pushes the nucleus to the side . Eventually , large drops of neighboring liver cells coalesce to form fatty cysts , in which the ...
... nucleus , however , still remaining in the center . Subsequently , the droplets merge , and one large drop pushes the nucleus to the side . Eventually , large drops of neighboring liver cells coalesce to form fatty cysts , in which the ...
Page 62
... nucleus , becoming pyknotic , eventually disappears . These cell rem- nants are then expelled from the liver plate and lie in the tissue spaces as acido- philic masses , the so - called " Councilman bodies " , named because of the fact ...
... nucleus , becoming pyknotic , eventually disappears . These cell rem- nants are then expelled from the liver plate and lie in the tissue spaces as acido- philic masses , the so - called " Councilman bodies " , named because of the fact ...
Page 86
... nucleus . The distended liver is smooth , glassy and pink - brown . The fat content is in most instances higher than normal . Excessive glycogen deposition may occur also in other organs , especially kidneys and heart . The pathogenesis ...
... nucleus . The distended liver is smooth , glassy and pink - brown . The fat content is in most instances higher than normal . Excessive glycogen deposition may occur also in other organs , especially kidneys and heart . The pathogenesis ...
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fatty fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall