The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
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Page 82
... produce jaundice , because the nonobstructed part of the liver takes over in a compensating fashion . The excretion of bile compo- nents other than the bile pigments , how- ever , may not be as readily compensated , and serum alkaline ...
... produce jaundice , because the nonobstructed part of the liver takes over in a compensating fashion . The excretion of bile compo- nents other than the bile pigments , how- ever , may not be as readily compensated , and serum alkaline ...
Page 125
... produce jaundice in at least 65 per cent of the cases . Ductal concretions are either muddy material or gravel or calculi , which may form directly in the ducts , e.g. , after cholecystectomy . But , in gen- eral , choledocholithiasis ...
... produce jaundice in at least 65 per cent of the cases . Ductal concretions are either muddy material or gravel or calculi , which may form directly in the ducts , e.g. , after cholecystectomy . But , in gen- eral , choledocholithiasis ...
Page 126
... produce acute cholecys- titis , varying from a diffuse inflammatory reaction of the mucosa to circumscribed ... produce jaun- dice . A pancreatic disease occasionally develops , either because of reflux from bile into the pancreas or ...
... produce acute cholecys- titis , varying from a diffuse inflammatory reaction of the mucosa to circumscribed ... produce jaun- dice . A pancreatic disease occasionally develops , either because of reflux from bile into the pancreas or ...
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Common terms and phrases
abdominal abnormal abscesses acid Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic CIBA cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts degeneration develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fatty fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior inflammatory intestinal intrahepatic jaundice Kupffer cells left hepatic lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatics MESENTERIC VEIN metabolism metastases mucosa necrosis Netter M.D. OCIBA nodes normal organ pancreatic duct pancreaticoduodenal papilla parenchyma patients peritoneal pigment portal hypertension portal triads portal vein posterior protein result right hepatic septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tract tumor urine urobilinogen usually vena cava vessels viral hepatitis wall