Bockus Gastroenterology: Liver |
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Page 2762
... increased ( usually ≤ 20 % ) ; secondary rise at 90-120 minutes Markedly reduced ( 0.9 0.4 mg / min ) Normal Normal or slightly increased total ; > 80 % as coproporphyrin - I Impaired biliary excretion of conjugated organic anions ...
... increased ( usually ≤ 20 % ) ; secondary rise at 90-120 minutes Markedly reduced ( 0.9 0.4 mg / min ) Normal Normal or slightly increased total ; > 80 % as coproporphyrin - I Impaired biliary excretion of conjugated organic anions ...
Page 2976
... increased amounts of collagen needs further study . Acetaldehyde has been incriminated in this process.271a 195a The accumulation of hepatic collagen dur- ing the development of cirrhosis could theo- retically be accomplished by ...
... increased amounts of collagen needs further study . Acetaldehyde has been incriminated in this process.271a 195a The accumulation of hepatic collagen dur- ing the development of cirrhosis could theo- retically be accomplished by ...
Page 2989
... increases hepatic oxygen consumption ; ATP- ase activity is increased in proportion to the increased oxygen consumption . When the increased oxygen demand is not satisfied by increased supplies , oxygen tension is re- duced and hepatic ...
... increases hepatic oxygen consumption ; ATP- ase activity is increased in proportion to the increased oxygen consumption . When the increased oxygen demand is not satisfied by increased supplies , oxygen tension is re- duced and hepatic ...
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Common terms and phrases
abnormalities acute hepatitis acute viral hepatitis agent albumin alcoholic hepatitis alcoholic liver disease amino acids Ann Intern antibody antigen ascites associated atitis Berk PD bile acids bile duct bilirubin Biochem bleeding cause cholangitis cholestasis cholestatic cholesterol chronic active hepatitis chronic hepatitis chronic liver disease cirrhotic Clin Invest clinical collagen concentration conjugated cytoplasm diagnosis drug effect encephalopathy Engl enzyme excretion fatty liver fibrosis fulminant hepatic function Gastroenterol Gastroenterology Gilbert's syndrome granulomas HBsAg heme hepa hepatic failure hepatitis B virus hepatocellular hepatocytes Hepatology histologic hyperbilirubinemia increased infection intrahepatic jaundice Lancet lesion Lieber lipid liver biopsy liver cell liver injury membrane microsomal necrosis non-A non-B hepatitis normal oxidation Pathol patients plasma Popper H portal hypertension portal tracts primary biliary cirrhosis protein rat liver renal Schaffner F Sherlock shunt sinusoidal sodium studies synthesis therapy tients tion tissue toxic transferase treatment unconjugated uptake usually varices vein viral hepatitis