Bockus Gastroenterology, Volume 5 |
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Page 2835
As indicated by the name, the lesion is located in the hepatic lobules, but
confluent or bridging necrosis and piecemeal necrosis are not part of the CLH
lesion. The terms persistent, unresolved, or prolonged viral hepatitis denote
similar lesions ...
As indicated by the name, the lesion is located in the hepatic lobules, but
confluent or bridging necrosis and piecemeal necrosis are not part of the CLH
lesion. The terms persistent, unresolved, or prolonged viral hepatitis denote
similar lesions ...
Page 2837
CAH with abnormal biliary epithelium has a worse prognosis than CAH without
this lesion, progressing more quickly and more frequently to cirrhosis.” CAH with
Confluent Hepatic Necrosis. In 1970, Boyer and Klatskin” described confluent ...
CAH with abnormal biliary epithelium has a worse prognosis than CAH without
this lesion, progressing more quickly and more frequently to cirrhosis.” CAH with
Confluent Hepatic Necrosis. In 1970, Boyer and Klatskin” described confluent ...
Page 3019
However, depending on the location of the cut, the lesion may also appear to be "
midzonal"”° and lesions of this type have been produced experimentally?” When
the hepatic acinus is severely injured, as in phospl'iOl'LiS poisoning, the ...
However, depending on the location of the cut, the lesion may also appear to be "
midzonal"”° and lesions of this type have been produced experimentally?” When
the hepatic acinus is severely injured, as in phospl'iOl'LiS poisoning, the ...
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Bockus gastroenterology, Volume 7 Henry L. Bockus,Jack Edward Berk,William S. Haubrich Snippet view - 1985 |
Common terms and phrases
abnormalities acute hepatitis acute viral hepatitis agent albumin alcoholic hepatitis alcoholic liver disease amino acids antibody antigen ascites associated atitis Berk PD bile acids bile duct bilirubin Biochem bleeding blood flow cause cholangitis cholestasis cholestatic cholesterol chronic active hepatitis chronic hepatitis chronic liver disease cirrhotic clinical collagen concentration conjugated cytoplasm develop diagnosis drug effect encephalopathy Engl enzyme excretion fatty liver fibrosis fluid function Gastroenterol Gastroenterology Gilbert's syndrome HBsAg heme hepa hepatocellular hepatocytes Hepatology histologic hyperbilirubinemia increased infection inflammatory intrahepatic jaundice Lancet lesion levels Lieber lipid liver biopsy liver cell liver injury lymphocytes membrane microsomal necrosis non-A non-B hepatitis normal oxidation pathogenesis Pathol patients plasma Popper H portal hypertension portal tracts primary biliary cirrhosis protein rat liver renal reported Schaffner F Sherlock shunt sinusoidal sodium studies synthesis therapy tients tion tissue toxic transferase treatment unconjugated uptake usually varices vein viral hepatitis