Bockus Gastroenterology: Liver |
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Page 3126
... sodium and water retention , plasma volume returns toward normal , portal pressure rises again , and ascites formation resumes . Normal plasma volume is never achieved because of continued ascites formation , and thus renal sodium ...
... sodium and water retention , plasma volume returns toward normal , portal pressure rises again , and ascites formation resumes . Normal plasma volume is never achieved because of continued ascites formation , and thus renal sodium ...
Page 3130
... Sodium Balance and Ascites . Basic to the management of ascites in liver disease is an understanding of sodium balance . Both as- cites and edema fluid are ultrafiltrates of plasma with a sodium content slightly less than plasma because ...
... Sodium Balance and Ascites . Basic to the management of ascites in liver disease is an understanding of sodium balance . Both as- cites and edema fluid are ultrafiltrates of plasma with a sodium content slightly less than plasma because ...
Page 3131
... sodium intake . A 24 - hour urinary sodium level below 5 mEq or a concentration below 5 mEq / liter indicates " tight " sodium retention and the need for potent diuretics . If the 24 - hour urinary so- dium level is neither very low nor ...
... sodium intake . A 24 - hour urinary sodium level below 5 mEq or a concentration below 5 mEq / liter indicates " tight " sodium retention and the need for potent diuretics . If the 24 - hour urinary so- dium level is neither very low nor ...
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Common terms and phrases
abnormalities acute hepatitis acute viral hepatitis agent albumin alcoholic hepatitis alcoholic liver disease amino acids Ann Intern antibody antigen ascites associated atitis Berk PD bile acids bile duct bilirubin Biochem bleeding cause cholangitis cholestasis cholestatic cholesterol chronic active hepatitis chronic hepatitis chronic liver disease cirrhotic Clin Invest clinical collagen concentration conjugated cytoplasm diagnosis drug effect encephalopathy Engl enzyme excretion fatty liver fibrosis fulminant hepatic function Gastroenterol Gastroenterology Gilbert's syndrome granulomas HBsAg heme hepa hepatic failure hepatitis B virus hepatocellular hepatocytes Hepatology histologic hyperbilirubinemia increased infection intrahepatic jaundice Lancet lesion Lieber lipid liver biopsy liver cell liver injury membrane microsomal necrosis non-A non-B hepatitis normal oxidation Pathol patients plasma Popper H portal hypertension portal tracts primary biliary cirrhosis protein rat liver renal Schaffner F Sherlock shunt sinusoidal sodium studies synthesis therapy tients tion tissue toxic transferase treatment unconjugated uptake usually varices vein viral hepatitis