The Ciba Collection of Medical Illustrations: Respiratory systemThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
From inside the book
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Page 35
... become the two main bronchi . As soon as the right bronchus appears , it is a little larger than the left one and tends to be more vertically oriented ( Plates 32 and 35 ) . These differences become more pronounced up to and after the ...
... become the two main bronchi . As soon as the right bronchus appears , it is a little larger than the left one and tends to be more vertically oriented ( Plates 32 and 35 ) . These differences become more pronounced up to and after the ...
Page 40
... becomes highly vascularized . During the sixth month the epithelium of the terminal sacs thins where it is in contact with a capillary ( Plate 39 ) . The epithelial cells become so thin when the alveoli fill with air that , before the ...
... becomes highly vascularized . During the sixth month the epithelium of the terminal sacs thins where it is in contact with a capillary ( Plate 39 ) . The epithelial cells become so thin when the alveoli fill with air that , before the ...
Page 113
... become infected . Bronchogenic cysts must be distin- guished from acquired bronchiectasis , which is more common in ... become very large without causing symptoms . However , in the subcarinal area they can cause pressure symptoms even ...
... become infected . Bronchogenic cysts must be distin- guished from acquired bronchiectasis , which is more common in ... become very large without causing symptoms . However , in the subcarinal area they can cause pressure symptoms even ...
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Common terms and phrases
abnormalities abscess acute airflow airway alveolar alveolar pressure alveoli aorta asthma basal biopsy blood flow breathing bron bronchial bronchial artery bronchitis bronchogenic bronchoscopy bronchus capillary carbon dioxide carcinoma cartilage cause cavity cells chest wall chronic CIBA clinical Continued cor pulmonale costal cough diagnosis diaphragm diffuse drainage dyspnea edema elastic recoil embolism emphysema esophagus expiration factor fibers fibrosis fluid gas exchange gland hypercapnia hypoventilation hypoxemia increased infection inhalation intercostal interstitial lateral left lung lesions lower lobe lung volume lymph nodes mechanical medial mediastinal mediastinum membrane nerve Netter CIBA Netter M.D. CIBA nodules normal obstruction occur oxygen patients peripheral pleural pressure pneumonia pneumothorax posterior pulmo pulmonary artery pulmonary embolism respiratory result right lung roentgenogram sarcoidosis SECTION IV PLATE segment sputum superior surface symptoms syndrome therapy thoracic tion tissue trachea tracheobronchial tube tuberculosis tumor upper lobe usually vascular vein vena cava venous ventilation vessels