The Ciba Collection of Medical Illustrations: Respiratory systemThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
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Page 71
Hyperventilation is seen in excessively anxious or apprehensive individuals , and
also occurs secondary to fever and following the ingestion of drugs such as
aspirin which act as respiratory stimulants . Certain disorders of the central
nervous ...
Hyperventilation is seen in excessively anxious or apprehensive individuals , and
also occurs secondary to fever and following the ingestion of drugs such as
aspirin which act as respiratory stimulants . Certain disorders of the central
nervous ...
Page 79
Abnormal Breathing Patterns Alveolar hyperventilation or hypoventilation usually
results from multiple rather than single causes . Hyperventilation occurring in
pulmonary edema is caused by hypoxemia and probably by stimulation of irritant
...
Abnormal Breathing Patterns Alveolar hyperventilation or hypoventilation usually
results from multiple rather than single causes . Hyperventilation occurring in
pulmonary edema is caused by hypoxemia and probably by stimulation of irritant
...
Page 290
Hypercapnia ( Paco , > 42 mm Hg ) is due to hypoventilation ; hypocapnia ( Paco
, < 38 mm Hg ) , to hyperventilation . Base excess is defined as zero for a blood
pH of 7.40 and a Paco , of 40 mm Hg . Theoretically it can be determined by ...
Hypercapnia ( Paco , > 42 mm Hg ) is due to hypoventilation ; hypocapnia ( Paco
, < 38 mm Hg ) , to hyperventilation . Base excess is defined as zero for a blood
pH of 7.40 and a Paco , of 40 mm Hg . Theoretically it can be determined by ...
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Common terms and phrases
abnormalities acute airway alveolar alveoli amphotericin appear artery associated asthma become blood body breathing bronchial bronchus capacity capillary carbon dioxide carcinoma cartilage cause cavity cells changes chest chronic CIBA clinical close common concentration contains Continued develop diagnosis diaphragm diffuse disease edema effect emphysema examination expiration factor flow fluid frequently function heart hyperventilation increased indicated infection inspiration intercostal interstitial involved lateral lesions less lobe lower lower lobe lung volume lymph major measured mechanical membrane muscle nerve Netter nodes normal obstruction occur organisms oxygen patients pattern PLATE pleural pneumonia position posterior present pressure produce pulmonary resistance respiratory response result SECTION IV PLATE seen segment severe side space sputum superior surface therapy thoracic thoracotomy tion tissue trachea treatment tube tumor upper lobe usually vein venous ventilation vessels volume wall x-ray