The Ciba Collection of Medical Illustrations: HeartThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
From inside the book
Results 1-3 of 42
Page 133
... anomaly is a persistence of the left superior vena cava . This vein , after being formed by the confluence of the left jugular and the subclavian veins , descends into the chest parallel to the right superior vena cava , anterior to the ...
... anomaly is a persistence of the left superior vena cava . This vein , after being formed by the confluence of the left jugular and the subclavian veins , descends into the chest parallel to the right superior vena cava , anterior to the ...
Page 143
... Anomaly of the Tricuspid Valve Ebstein's anomaly of the tricuspid valve , as an isolated malformation , is less common than tricuspid atresia . It is , how- ever , of considerable clinical importance , since most patients reach ...
... Anomaly of the Tricuspid Valve Ebstein's anomaly of the tricuspid valve , as an isolated malformation , is less common than tricuspid atresia . It is , how- ever , of considerable clinical importance , since most patients reach ...
Page 144
... ANOMALY CLAMP HAS BEEN MOMENTARILY LOOSENED TO PERMIT INTERATRIAL SEPTUM TO. R. ATRIUM R. VENTRICLE SECTION OF R. ATRIO- VENTRICULAR JUNCTION IN NORMAL HEART & Netter OCIBA SEVERE FORM OF EBSTEIN'S ANOMALY SECTION IV - PLATE 12 ANOMALIES ...
... ANOMALY CLAMP HAS BEEN MOMENTARILY LOOSENED TO PERMIT INTERATRIAL SEPTUM TO. R. ATRIUM R. VENTRICLE SECTION OF R. ATRIO- VENTRICULAR JUNCTION IN NORMAL HEART & Netter OCIBA SEVERE FORM OF EBSTEIN'S ANOMALY SECTION IV - PLATE 12 ANOMALIES ...
Other editions - View all
Common terms and phrases
A-V node aneurysm anomaly aortic arch aortic valve apex arrhythmias ascending aorta atrial atrioventricular block blood flow branch bundle cardiac output cardinal veins catheter cause cells chordae tendineae clinical common Continued conus coronary artery CUSP depolarization develop diastolic digitalis dilatation drugs edema electrocardiogram embryo endocardial enlargement fibers fibrillation heart failure hypertension increase INFERIOR VENA CAVA leads left atrium left ventricle left ventricular lesions lung M.D. OCIBA medial membrane mitral insufficiency mitral stenosis mitral valve murmur myocardial infarction myocardium NERVE Netter CIBA Netter M.D. normal occur oxygen pacemaker PAPILLARY MUSCLE patients pericardial pericardium peripheral portion posterior pressure PULMONARY ARTERY PULMONARY TRUNK PULMONARY VEINS QRS complex renal result rheumatic right atrium right ventricle right ventricular S-T segments SECTION II-PLATE SECTION V-PLATE shunt sound SUPERIOR VENA CAVA suture sympathetic systolic tachycardia thoracic tion tissue tricular tricuspid valve truncus usually vagal valvular vascular venous ventricular septal defect vessels wall waves