The Ciba Collection of Medical Illustrations: HeartThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
From inside the book
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Page 84
... cause , a most im- portant favoring element is the rapid shift of a large mass of blood from the periphery to the lungs as a result of systemic vasoconstriction ( narrowing of both the arterioles and the ven- ules ) . This is often caused ...
... cause , a most im- portant favoring element is the rapid shift of a large mass of blood from the periphery to the lungs as a result of systemic vasoconstriction ( narrowing of both the arterioles and the ven- ules ) . This is often caused ...
Page 106
... cause a general sympathetic discharge . The involvement of the cardiovascular system is prominent , and a precipitous increase in cardiac output and rise in blood pressure may result in fatal cerebral hemorrhage . Paradoxically , MAO ...
... cause a general sympathetic discharge . The involvement of the cardiovascular system is prominent , and a precipitous increase in cardiac output and rise in blood pressure may result in fatal cerebral hemorrhage . Paradoxically , MAO ...
Page 181
... cause bacterial endocarditis . The skin , especially in infants with eczematous lesions , may per- mit entry of highly virulent staphylococci . From infection of the upper respiratory tract , B - hemolytic streptococcus is an occasional ...
... cause bacterial endocarditis . The skin , especially in infants with eczematous lesions , may per- mit entry of highly virulent staphylococci . From infection of the upper respiratory tract , B - hemolytic streptococcus is an occasional ...
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Common terms and phrases
A-V node aneurysm anomaly aortic arch aortic valve apex arrhythmias ascending aorta atrial atrioventricular block blood flow branch bundle cardiac output cardinal veins catheter cause cells chordae tendineae clinical common Continued conus coronary artery CUSP depolarization develop diastolic digitalis dilatation drugs edema electrocardiogram embryo endocardial enlargement fibers fibrillation heart failure hypertension increase INFERIOR VENA CAVA leads left atrium left ventricle left ventricular lesions lung M.D. OCIBA medial membrane mitral insufficiency mitral stenosis mitral valve murmur myocardial infarction myocardium NERVE Netter CIBA Netter M.D. normal occur oxygen pacemaker PAPILLARY MUSCLE patients pericardial pericardium peripheral portion posterior pressure PULMONARY ARTERY PULMONARY TRUNK PULMONARY VEINS QRS complex renal result rheumatic right atrium right ventricle right ventricular S-T segments SECTION II-PLATE SECTION V-PLATE shunt sound SUPERIOR VENA CAVA suture sympathetic systolic tachycardia thoracic tion tissue tricular tricuspid valve truncus usually vagal valvular vascular venous ventricular septal defect vessels wall waves