The Ciba Collection of Medical Illustrations: HeartThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
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Page 39
... ( INCREASED O2 SUPPLY AND METABOLITE REMOVAL ) -INCREASED MYOCARDIAL METABOLISM- FRANK - STARLING EFFECT INCREASED VENOUS RETURN EFFECTS OF RESTING TENSION , CORONARY BLOOD FLOW , AND NOREPINEPHRINE ON MYOCARDIAL CONTRACTION RESTING ...
... ( INCREASED O2 SUPPLY AND METABOLITE REMOVAL ) -INCREASED MYOCARDIAL METABOLISM- FRANK - STARLING EFFECT INCREASED VENOUS RETURN EFFECTS OF RESTING TENSION , CORONARY BLOOD FLOW , AND NOREPINEPHRINE ON MYOCARDIAL CONTRACTION RESTING ...
Page 104
... INCREASED EPINEPHRINE HEART : B - RECEPTORS STRONGLY STIMULATED RATE INCREASED FORCE OF CONTRACTION INCREASED CORONARY PERFUSION INCREASED AUTOMATICITY INCREASED—— VASCULAR BED : -a - RECEPTORS MILDLY STIMULATED AT SOME SITES , STRONGLY ...
... INCREASED EPINEPHRINE HEART : B - RECEPTORS STRONGLY STIMULATED RATE INCREASED FORCE OF CONTRACTION INCREASED CORONARY PERFUSION INCREASED AUTOMATICITY INCREASED—— VASCULAR BED : -a - RECEPTORS MILDLY STIMULATED AT SOME SITES , STRONGLY ...
Page 245
... INCREASED BLOOD VELOCITY INCREASED ( CIRCULATION TIME DECREASED ) HEART RATE INCREASED HEART IN HYPERTHYROIDISM The striking relationship between thy- rotoxicosis and the circulation was recog- nized as early as 1786 by Parry , who ...
... INCREASED BLOOD VELOCITY INCREASED ( CIRCULATION TIME DECREASED ) HEART RATE INCREASED HEART IN HYPERTHYROIDISM The striking relationship between thy- rotoxicosis and the circulation was recog- nized as early as 1786 by Parry , who ...
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Common terms and phrases
A-V node aneurysm anomaly aortic arch aortic valve apex arrhythmias ascending aorta atrial atrioventricular block blood flow branch bundle cardiac output cardinal veins catheter cause cells chordae tendineae clinical common Continued conus coronary artery CUSP depolarization develop diastolic digitalis dilatation drugs edema electrocardiogram embryo endocardial enlargement fibers fibrillation heart failure hypertension increase INFERIOR VENA CAVA leads left atrium left ventricle left ventricular lesions lung M.D. OCIBA medial membrane mitral insufficiency mitral stenosis mitral valve murmur myocardial infarction myocardium NERVE Netter CIBA Netter M.D. normal occur oxygen pacemaker PAPILLARY MUSCLE patients pericardial pericardium peripheral portion posterior pressure PULMONARY ARTERY PULMONARY TRUNK PULMONARY VEINS QRS complex renal result rheumatic right atrium right ventricle right ventricular S-T segments SECTION II-PLATE SECTION V-PLATE shunt sound SUPERIOR VENA CAVA suture sympathetic systolic tachycardia thoracic tion tissue tricular tricuspid valve truncus usually vagal valvular vascular venous ventricular septal defect vessels wall waves