The Ciba Collection of Medical Illustrations: HeartThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
From inside the book
Results 1-3 of 53
Page 95
... PERIPHERAL RESISTANCE BASIC THERAPY INCREASED FIRST : CAUSATIVE CONDITION ? VASOCONSTRICTORS IN GENERAL ? VARIABLE ... peripheral hypoxia may result , later , in dilatation and an increased permeability of the peripheral vascular bed ...
... PERIPHERAL RESISTANCE BASIC THERAPY INCREASED FIRST : CAUSATIVE CONDITION ? VASOCONSTRICTORS IN GENERAL ? VARIABLE ... peripheral hypoxia may result , later , in dilatation and an increased permeability of the peripheral vascular bed ...
Page 103
... peripheral resistance and hence a marked increase in systolic , diastolic , and mean arterial pressure due to peripheral vasoconstriction . Com- pensatory reflexes , arising from the carotid sinus and the aortic arch , activate the ...
... peripheral resistance and hence a marked increase in systolic , diastolic , and mean arterial pressure due to peripheral vasoconstriction . Com- pensatory reflexes , arising from the carotid sinus and the aortic arch , activate the ...
Page 104
... PERIPHERAL RESISTANCE PERIPHERAL◅ BLOOD FLOW DECREASED INCREASED EPINEPHRINE HEART : B - RECEPTORS STRONGLY STIMULATED RATE INCREASED FORCE OF CONTRACTION INCREASED CORONARY PERFUSION INCREASED AUTOMATICITY INCREASED—— VASCULAR BED ...
... PERIPHERAL RESISTANCE PERIPHERAL◅ BLOOD FLOW DECREASED INCREASED EPINEPHRINE HEART : B - RECEPTORS STRONGLY STIMULATED RATE INCREASED FORCE OF CONTRACTION INCREASED CORONARY PERFUSION INCREASED AUTOMATICITY INCREASED—— VASCULAR BED ...
Other editions - View all
Common terms and phrases
A-V node aneurysm anomaly aortic arch aortic valve apex arrhythmias ascending aorta atrial atrioventricular block blood flow branch bundle cardiac output cardinal veins catheter cause cells chordae tendineae clinical common Continued conus coronary artery CUSP depolarization develop diastolic digitalis dilatation drugs edema electrocardiogram embryo endocardial enlargement fibers fibrillation heart failure hypertension increase INFERIOR VENA CAVA leads left atrium left ventricle left ventricular lesions lung M.D. OCIBA medial membrane mitral insufficiency mitral stenosis mitral valve murmur myocardial infarction myocardium NERVE Netter CIBA Netter M.D. normal occur oxygen pacemaker PAPILLARY MUSCLE patients pericardial pericardium peripheral portion posterior pressure PULMONARY ARTERY PULMONARY TRUNK PULMONARY VEINS QRS complex renal result rheumatic right atrium right ventricle right ventricular S-T segments SECTION II-PLATE SECTION V-PLATE shunt sound SUPERIOR VENA CAVA suture sympathetic systolic tachycardia thoracic tion tissue tricular tricuspid valve truncus usually vagal valvular vascular venous ventricular septal defect vessels wall waves