The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
From inside the book
Results 1-3 of 29
Page 68
... lobular and nodular paren- chyma is also a stimulus for central necrosis as well as peripheral inflammation , both of which , in turn , provoke septa formation and separation of single or small groups of cells and , therewith ...
... lobular and nodular paren- chyma is also a stimulus for central necrosis as well as peripheral inflammation , both of which , in turn , provoke septa formation and separation of single or small groups of cells and , therewith ...
Page 94
... lobular architecture may be exaggerated , with dark - red depressed CUT SURFACE : ACUTELY CONGESTED , " SPLEENLIKE " LOW MAGNIFICATION : MASSIVE NECROSIS , INFILTRATION OF LOBULES AND PORTAL AREAS , BILE DUCT PROLIFERATION and enlarged ...
... lobular architecture may be exaggerated , with dark - red depressed CUT SURFACE : ACUTELY CONGESTED , " SPLEENLIKE " LOW MAGNIFICATION : MASSIVE NECROSIS , INFILTRATION OF LOBULES AND PORTAL AREAS , BILE DUCT PROLIFERATION and enlarged ...
Page 106
... lobular markings are far more distinct than usual . On closer inspection the zones around the central veins appear dark red and depressed and stand out distinctly against the intermediate and peripheral zones , which , sometimes ...
... lobular markings are far more distinct than usual . On closer inspection the zones around the central veins appear dark red and depressed and stand out distinctly against the intermediate and peripheral zones , which , sometimes ...
Other editions - View all
Common terms and phrases
abdominal abnormal abscesses acid acute pancreatitis Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches capillaries carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fatty fibers fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior intestinal intrahepatic jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatic M.D. CIBA metabolism metastases mucosa necrosis nerves Netter M.D. OCIBA nodes normal organ pancreatic duct papilla parenchyma patients peritoneal phosphatase pigment portal hypertension portal triads portal vein posterior protein result right hepatic septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tumor urine urobilinogen usually vena cava vessels viral hepatitis wall