The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
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Page 52
... PRESSURE DROPS kidney and adrenal compete for space would create pressure difficulties were it not for the gallbladder's ability to concentrate the liver bile from four- to ten- fold . The gallbladder mucosa can reabsorb water and salts ...
... PRESSURE DROPS kidney and adrenal compete for space would create pressure difficulties were it not for the gallbladder's ability to concentrate the liver bile from four- to ten- fold . The gallbladder mucosa can reabsorb water and salts ...
Page 69
... pressure , being at least partly transmitted into the portal vein branches by the anastomoses in the septa , increases the pressure in the portal vein . The arteriovenous anastomoses are , thus , the second main cause of portal hyper ...
... pressure , being at least partly transmitted into the portal vein branches by the anastomoses in the septa , increases the pressure in the portal vein . The arteriovenous anastomoses are , thus , the second main cause of portal hyper ...
Page 74
... pressure is higher than the oncotic pressure , is reabsorbed as interstitial fluid by the blood in the venous capillary limb , where the oncotic pressure exceeds the blood pressure . Disturbance of this mechanism results in accumulation ...
... pressure is higher than the oncotic pressure , is reabsorbed as interstitial fluid by the blood in the venous capillary limb , where the oncotic pressure exceeds the blood pressure . Disturbance of this mechanism results in accumulation ...
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Common terms and phrases
abdominal abnormal abscesses acid acute pancreatitis Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches capillaries carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fatty fibers fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior intestinal intrahepatic jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatic M.D. CIBA metabolism metastases mucosa necrosis nerves Netter M.D. OCIBA nodes normal organ pancreatic duct papilla parenchyma patients peritoneal phosphatase pigment portal hypertension portal triads portal vein posterior protein result right hepatic septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tumor urine urobilinogen usually vena cava vessels viral hepatitis wall