The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 3, Part 3Ciba Pharmaceutical Products, 1957 - Human anatomy |
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Page 49
... Urinary STOOL LIGHT # Netter M.D CIBA BOWEL urobilinogen excretion varies because of the inconstant flow of ... urinary urobilinogen is increased , owing to the liver cell injury which interferes with the enterohepatic circulation ...
... Urinary STOOL LIGHT # Netter M.D CIBA BOWEL urobilinogen excretion varies because of the inconstant flow of ... urinary urobilinogen is increased , owing to the liver cell injury which interferes with the enterohepatic circulation ...
Page 51
... urinary uro- bilinogen and Bromsulphalein reten- tion may disclose evidence of damage otherwise not available . However , a nor- mal outcome of these tests does not exclude liver cell damage , and the high incidence of false with one or ...
... urinary uro- bilinogen and Bromsulphalein reten- tion may disclose evidence of damage otherwise not available . However , a nor- mal outcome of these tests does not exclude liver cell damage , and the high incidence of false with one or ...
Page 56
... urinary amylase . Indeed , high excretion in the urine may persist for 24 hours after the blood amylase concen- tration has receded toward normal levels , and this persistence of urinary amylase # . Netter M.D. OCIBA excretion may serve ...
... urinary amylase . Indeed , high excretion in the urine may persist for 24 hours after the blood amylase concen- tration has receded toward normal levels , and this persistence of urinary amylase # . Netter M.D. OCIBA excretion may serve ...
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Common terms and phrases
abdominal abnormal abscesses acid acute pancreatitis Amer amylase anastomoses appear ascites become biliary obstruction bilirubin biopsy bladder blood branches capillaries carcinoma cause celiac cent cholangioles cholecystitis cholesterol chronic cirrhosis clinical common bile duct common hepatic connective tissue cystic artery cystic duct cysts develop diagnosis dilated duodenal duodenum enlarged enzymes esophageal varices excretion fatty fibers fibrosis fistula formation frequently function gallbladder gastric gland glycogen hemorrhage hepatic artery hepatic duct hepatic tests hepatic vein histologic infection inferior intestinal intrahepatic jaundice Kupffer cells lesions ligament liver cell plates liver disease lobe lobular lobule lymph lymphatic M.D. CIBA metabolism metastases mucosa necrosis nerves Netter M.D. OCIBA nodes normal organ pancreatic duct papilla parenchyma patients peritoneal phosphatase pigment portal hypertension portal triads portal vein posterior protein result right hepatic septa serum sinusoids sphincter spleen splenic stage stones SUPERIOR MESENTERIC surface surgical tion tumor urine urobilinogen usually vena cava vessels viral hepatitis wall