Bockus Gastroenterology, Volume 2 |
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Page 864
... parietal cells and thus may be considered multipotential . 52 , 53 The chief cells secrete pepsinogen , a proen- zyme that when activated by exposure to acid becomes the potently proteolytic en- zyme pepsin . The chief cells are located ...
... parietal cells and thus may be considered multipotential . 52 , 53 The chief cells secrete pepsinogen , a proen- zyme that when activated by exposure to acid becomes the potently proteolytic en- zyme pepsin . The chief cells are located ...
Page 903
... parietal cell changes dramatically during secretion , and intrinsic factor shares the general properties of exocrine protein se- cretion . Cellular Physiology of Gastric Secretion Parietal Cell Secretion Isolated Parietal Cells . The ...
... parietal cell changes dramatically during secretion , and intrinsic factor shares the general properties of exocrine protein se- cretion . Cellular Physiology of Gastric Secretion Parietal Cell Secretion Isolated Parietal Cells . The ...
Page 905
... parietal cells and stimulates the accumulation of ami- nopyrine with a potency equal to gastrin . Summary . Isolated parietal or oxyntic cells respond to secretagogues by accumulating acid in their cana- liculi , increasing oxygen ...
... parietal cells and stimulates the accumulation of ami- nopyrine with a potency equal to gastrin . Summary . Isolated parietal or oxyntic cells respond to secretagogues by accumulating acid in their cana- liculi , increasing oxygen ...
Contents
Motor Disorders of the Esophagus | 690 |
Hernias Hiatal Traumatic | 715 |
Reflux Esophagitis | 757 |
Copyright | |
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Common terms and phrases
abdominal abnormal achalasia acid output acute anemia Ann Surg antacid antral antrum aspirin associated atrophic gastritis barium benign biopsy bleeding blood carcinoma cause chronic cimetidine Clin clinical complications diagnosis dilatation diverticulum dose drugs duodenal ulcer duodenal ulcer patients duodenum dysphagia effect endoscopic epithelial epithelium erosions esoph esophageal sphincter factors fistula frequency function fundic gastrectomy gastric acid gastric and duodenal gastric emptying gastric mucosa gastric secretion gastric ulcer gastric vagotomy Gastroenterology gastroesophageal gastrointestinal tract glands Hâ‚‚ healing hemorrhage hiatal hernia histamine increased ingestion inhibition intestinal lesions lower esophageal lumen malignant meal muscle normal obstruction occur pain pancreatic parietal cells patients with duodenal peptic ulcer disease perforation postoperative prostaglandins pyloric radiologic ranitidine recurrent ulcer reflux esophagitis reported resection response Scand J Gastroenterol secretory serum gastrin stimulation stomach stricture sucralfate surgery surgical symptoms syndrome therapy thoracic tients tion tissue treatment tumor vagal vagotomy