Bockus Gastroenterology, Volume 2 |
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Page 833
... Response rates vary widely but in most randomized studies of 15 patients or more , response rates range from 15 % to 20 % . Unfortunately , even when a patient experiences a response to one of these drugs , the duration of tumor ...
... Response rates vary widely but in most randomized studies of 15 patients or more , response rates range from 15 % to 20 % . Unfortunately , even when a patient experiences a response to one of these drugs , the duration of tumor ...
Page 906
... response to picoprazole ( H 149/94 ) against histamine , CAMP , and potassium stimulated acid secretion . Acid output is determined as the accumulation of 1aC - aminopyrine in the canaliculi . ( From Fellenius E et al . Am J Physiol ...
... response to picoprazole ( H 149/94 ) against histamine , CAMP , and potassium stimulated acid secretion . Acid output is determined as the accumulation of 1aC - aminopyrine in the canaliculi . ( From Fellenius E et al . Am J Physiol ...
Page 911
... response to carbachol . Changes in pH from 8.0 to 6.7 caused small increases in the release of pepsinogen in response to isoproterenol , carbachol , and CCK - OP ; large increases followed 8 - bromoinosine cAMP ( 8BrcAMP ) and Forskolin ...
... response to carbachol . Changes in pH from 8.0 to 6.7 caused small increases in the release of pepsinogen in response to isoproterenol , carbachol , and CCK - OP ; large increases followed 8 - bromoinosine cAMP ( 8BrcAMP ) and Forskolin ...
Contents
Motor Disorders of the Esophagus | 690 |
Hernias Hiatal Traumatic | 715 |
Reflux Esophagitis | 757 |
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abdominal abnormal achalasia acid output acute anemia Ann Surg antacid antral antrum aspirin associated atrophic gastritis barium benign biopsy bleeding blood carcinoma cause chronic cimetidine Clin clinical complications diagnosis dilatation diverticulum dose drugs duodenal ulcer duodenal ulcer patients duodenum dysphagia effect endoscopic epithelial epithelium erosions esoph esophageal sphincter factors fistula frequency function fundic gastrectomy gastric acid gastric and duodenal gastric emptying gastric mucosa gastric secretion gastric ulcer gastric vagotomy Gastroenterology gastroesophageal gastrointestinal tract glands Hâ‚‚ healing hemorrhage hiatal hernia histamine increased ingestion inhibition intestinal lesions lower esophageal lumen malignant meal muscle normal obstruction occur pain pancreatic parietal cells patients with duodenal peptic ulcer disease perforation postoperative prostaglandins pyloric radiologic ranitidine recurrent ulcer reflux esophagitis reported resection response Scand J Gastroenterol secretory serum gastrin stimulation stomach stricture sucralfate surgery surgical symptoms syndrome therapy thoracic tients tion tissue treatment tumor vagal vagotomy