The Ciba Collection of Medical Illustrations: Respiratory systemThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
From inside the book
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Page 35
... become the two main bronchi . As soon as the right bronchus appears , it is a little larger than the left one and tends to be more vertically oriented ( Plates 32 and 35 ) . These differences become more pronounced up to and after the ...
... become the two main bronchi . As soon as the right bronchus appears , it is a little larger than the left one and tends to be more vertically oriented ( Plates 32 and 35 ) . These differences become more pronounced up to and after the ...
Page 40
... becomes highly vascularized . During the sixth month the epithelium of the terminal sacs thins where it is in contact with a capillary ( Plate 39 ) . The epithelial cells become so thin when the alveoli fill with air that , before the ...
... becomes highly vascularized . During the sixth month the epithelium of the terminal sacs thins where it is in contact with a capillary ( Plate 39 ) . The epithelial cells become so thin when the alveoli fill with air that , before the ...
Page 113
... become infected . Bronchogenic cysts must be distin- guished from acquired bronchiectasis , which is more common in ... become very large without causing symptoms . However , in the subcarinal area they can cause pressure symptoms even ...
... become infected . Bronchogenic cysts must be distin- guished from acquired bronchiectasis , which is more common in ... become very large without causing symptoms . However , in the subcarinal area they can cause pressure symptoms even ...
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Common terms and phrases
abnormalities acute airflow airway alveolar alveoli aorta aortic asthma basal blood flow brachiocephalic breathing bron bronchial artery bronchogenic capillary carbon dioxide carcinoma cartilage cause caveola cavity cells cervical chest wall chronic CIBA clinical Continued cor pulmonale costal cough diagnosis diaphragm diaphragmatic diffuse disease drainage duct dyspnea edema embolism emphysema epithelium esophagus expiration fibers fibrosis fluid gas exchange gland hypoventilation hypoxemia increased infection inferior intercostal interstitial lateral left lung lesions lower lobe lung volume lymph nodes main bronchus medial mediastinal mediastinum membrane nerve Netter CIBA Netter M.D. CIBA normal obstruction occur oxygen parietal pleura patients pericardial phrenic pleural pressure pneumonia pneumothorax posterior pulmo pulmonary artery pulmonary embolism Respir respiratory result right lung roentgenogram SECTION IV PLATE segment smooth muscle sputum superior surface syndrome therapy thoracic vertebra tion tissue trachea tracheobronchial tube tuberculosis tumor upper lobe usually vascular vein vena cava venous ventilation vessels