The Ciba Collection of Medical Illustrations: Respiratory systemThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
From inside the book
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Page 30
... frequently consist only of the extremely flat- tened extensions of endothelial cells and type I alveolar cells . In other regions the wall contains such cell types as smooth muscle cells , pericytes , fibroblasts and occasional plasma ...
... frequently consist only of the extremely flat- tened extensions of endothelial cells and type I alveolar cells . In other regions the wall contains such cell types as smooth muscle cells , pericytes , fibroblasts and occasional plasma ...
Page 171
... frequently confused with primary or metastatic malignancies of the lung . Endobronchial lesions , on the other hand , may not be seen as distinct x - ray shadows but they may induce cough , sputum production , hemop- tysis or localized ...
... frequently confused with primary or metastatic malignancies of the lung . Endobronchial lesions , on the other hand , may not be seen as distinct x - ray shadows but they may induce cough , sputum production , hemop- tysis or localized ...
Page 173
... frequently occurs from 30 to 50 years after the initial exposure to asbestos . The mean age at onset of symptoms is around 60 years . The initial symptoms are notoriously vague and are often disregarded by the physician ; the patient ...
... frequently occurs from 30 to 50 years after the initial exposure to asbestos . The mean age at onset of symptoms is around 60 years . The initial symptoms are notoriously vague and are often disregarded by the physician ; the patient ...
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Common terms and phrases
abnormalities acute airflow airway alveolar alveoli aorta aortic asthma basal blood flow brachiocephalic breathing bron bronchial artery bronchogenic capillary carbon dioxide carcinoma cartilage cause caveola cavity cells cervical chest wall chronic CIBA clinical Continued cor pulmonale costal cough diagnosis diaphragm diaphragmatic diffuse disease drainage duct dyspnea edema embolism emphysema epithelium esophagus expiration fibers fibrosis fluid gas exchange gland hypoventilation hypoxemia increased infection inferior intercostal interstitial lateral left lung lesions lower lobe lung volume lymph nodes main bronchus medial mediastinal mediastinum membrane nerve Netter CIBA Netter M.D. CIBA normal obstruction occur oxygen parietal pleura patients pericardial phrenic pleural pressure pneumonia pneumothorax posterior pulmo pulmonary artery pulmonary embolism Respir respiratory result right lung roentgenogram SECTION IV PLATE segment smooth muscle sputum superior surface syndrome therapy thoracic vertebra tion tissue trachea tracheobronchial tube tuberculosis tumor upper lobe usually vascular vein vena cava venous ventilation vessels