The Ciba Collection of Medical Illustrations: Respiratory systemThe most critically acclaimed of all of Dr. Frank H. Netter's works, this fully illustrated single book from the 8-volume/13-book reference collection includes: hundreds of world-renowned illustrations by Frank H. Netter, MD; informative text by recognized medical experts; anatomy, physiology, and pathology; and diagnostic and surgical procedures. |
From inside the book
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Page 71
... hyper- ventilation . The excessive output of carbon dioxide leads to hypocapnia , which elevates the pH . Hyperventilation is seen in excessively anx- ious or apprehensive individuals , and also occurs secondary to fever and following ...
... hyper- ventilation . The excessive output of carbon dioxide leads to hypocapnia , which elevates the pH . Hyperventilation is seen in excessively anx- ious or apprehensive individuals , and also occurs secondary to fever and following ...
Page 79
... hyperventilation or hypoventilation usually results from multiple rather than single causes . Hyperventilation occurring in pulmonary edema is caused by hypoxemia and probably by stimulation of irritant and J receptors in the lung ...
... hyperventilation or hypoventilation usually results from multiple rather than single causes . Hyperventilation occurring in pulmonary edema is caused by hypoxemia and probably by stimulation of irritant and J receptors in the lung ...
Page 290
... hyperventilation . Base excess is defined as zero for a blood pH of 7.40 and a Paco , of 40 mm Hg . Theoretically it can be determined by titration of the blood to a pH of 7.40 at a Paco , of 40 mm Hg and a temperature of 38 ° C , but ...
... hyperventilation . Base excess is defined as zero for a blood pH of 7.40 and a Paco , of 40 mm Hg . Theoretically it can be determined by titration of the blood to a pH of 7.40 at a Paco , of 40 mm Hg and a temperature of 38 ° C , but ...
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Common terms and phrases
abnormalities acute airflow airway alveolar alveoli aorta aortic asthma basal blood flow brachiocephalic breathing bron bronchial artery bronchogenic capillary carbon dioxide carcinoma cartilage cause caveola cavity cells cervical chest wall chronic CIBA clinical Continued cor pulmonale costal cough diagnosis diaphragm diaphragmatic diffuse disease drainage duct dyspnea edema embolism emphysema epithelium esophagus expiration fibers fibrosis fluid gas exchange gland hypoventilation hypoxemia increased infection inferior intercostal interstitial lateral left lung lesions lower lobe lung volume lymph nodes main bronchus medial mediastinal mediastinum membrane nerve Netter CIBA Netter M.D. CIBA normal obstruction occur oxygen parietal pleura patients pericardial phrenic pleural pressure pneumonia pneumothorax posterior pulmo pulmonary artery pulmonary embolism Respir respiratory result right lung roentgenogram SECTION IV PLATE segment smooth muscle sputum superior surface syndrome therapy thoracic vertebra tion tissue trachea tracheobronchial tube tuberculosis tumor upper lobe usually vascular vein vena cava venous ventilation vessels