Bockus Gastroenterology, Volume 1 |
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Page 324
... factor XII that makes it more susceptible to cleavage by kallikrein is thought to be involved , and the reaction is accelerated in a nonenzymatic fashion by high molecular weight kininogen . Once factor XII is activated , it in turn ...
... factor XII that makes it more susceptible to cleavage by kallikrein is thought to be involved , and the reaction is accelerated in a nonenzymatic fashion by high molecular weight kininogen . Once factor XII is activated , it in turn ...
Page 325
... factor V. Factor V is analogous to factor VIII in that it has no protease activity of its own but greatly accelerates the proteo- lytic activity of factor Xa . Like factor VIII , it too can be activated by catalytic amounts of thrombin ...
... factor V. Factor V is analogous to factor VIII in that it has no protease activity of its own but greatly accelerates the proteo- lytic activity of factor Xa . Like factor VIII , it too can be activated by catalytic amounts of thrombin ...
Page 328
... factors XII , IX , and VIII ) , or a prothrombin time ( for factors VII , X , V , and II ) . In the case of factor X , Russell's viper venom , a direct activator of this clotting factor , may be substituted for tissue factor . RUSSELL'S ...
... factors XII , IX , and VIII ) , or a prothrombin time ( for factors VII , X , V , and II ) . In the case of factor X , Russell's viper venom , a direct activator of this clotting factor , may be substituted for tissue factor . RUSSELL'S ...
Contents
SYMPTOMATOLOGY | 24 |
Abdominal Scout Film Assessment | 32 |
Abdominal Pain | 36 |
Copyright | |
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abdominal pain abnormal abscess acid acute abdomen aerophagia anemia aneurysm anorexia artery ascitic fluid associated bacterial barium bile biliary bilirubin blood carcinoma cause cells Chapter cholecystitis chronic cirrhosis Clin clinical colitis colon constipation Crohn's disease deficiency detected diagnosis diarrhea distention drugs duct duodenal duodenum dysphagia endoscopy enema Engl esophageal factor fecal flatus frequently gallbladder gastric Gastroenterology gastrointes gastrointestinal bleeding gastrointestinal tract hematemesis hemorrhage hepatic increased infarction infection inflammatory ingestion irritable jaundice lesions liver M.D. Professor malabsorption mechanism Medical melena mesenteric mucosa nausea normal obstruction occur oral organic palpation pancreatic patients peptic ulcer peritonitis plasma platelet portal portal hypertension present pressure Professor of Medicine rare rectal rectum reflex renal result rupture School of Medicine serum skin small bowel small intestine sphincter splenic stomach stool studies Surg surgery surgical swallowing symptoms syndrome tenderness therapy tients tion tumors upper gastrointestinal usually varices vascular visceral vomiting