The Ciba Collection of Medical Illustrations: A Compilation of Pathological and Anatomical Paintings, Volume 4Ciba Pharmaceutical Products, 1965 - Human anatomy |
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Page 131
... ovaries and testes yield some of these , notably dehydroepiandrosterone . Yet the sixtyfold more potent testosterone is not a 17 - ketosteroid . In the ovary much of the testosterone is normally trans- formed into estrogen . When the ...
... ovaries and testes yield some of these , notably dehydroepiandrosterone . Yet the sixtyfold more potent testosterone is not a 17 - ketosteroid . In the ovary much of the testosterone is normally trans- formed into estrogen . When the ...
Page 133
... ovaries are small structures made up entirely of primordial follicles , disclosing no elements capable of producing estrogens . In the decade of childhood , from the postnatal recessional changes to the time of puberty , the ovaries ...
... ovaries are small structures made up entirely of primordial follicles , disclosing no elements capable of producing estrogens . In the decade of childhood , from the postnatal recessional changes to the time of puberty , the ovaries ...
Page 225
... OVARY The adrenal cortex contributes very weak andro- gens , collectively known as 17 - ketosteroids ( 17 - KS ) . Their anabolic effect is important in females , who obtain little androgen from their ovaries . Small amounts of ...
... OVARY The adrenal cortex contributes very weak andro- gens , collectively known as 17 - ketosteroids ( 17 - KS ) . Their anabolic effect is important in females , who obtain little androgen from their ovaries . Small amounts of ...
Contents
NUMBER | 1 |
PLATE | 3 |
Relationship of the Pituitary Gland to | 7 |
Copyright | |
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17-ketosteroids abnormal acromegaly ACTH activity adenohypophysis adenoma adrenal cortical adult aldosterone amino acids androgens ARTERY atrophy basophil bone calcium cancer capillary carcinoma cause cells cent chiasm cholesterol chromophobe CHROMOPHOBE ADENOMA chromosome CIBA clinical cortex CORTICOIDS cortisol Cushing's Cushing's syndrome decrease deficiency diabetes insipidus differentiation disease duct effect elevated enlarged enzyme estrogen excessive excretion exophthalmos fatty acids female follicles function glucose glycogen goiter gonadal gonadotropins hyperplasia hyperthyroidism hypoglycemia hypophysial hypothalamus increased inhibition insulin iodine kidney lesions liver lobe M.D. OCIBA male metabolism molecule muscle myxedema nerve Netter M.D. normal obesity occur ovaries parathyroid patients phosphate pineal pituitary gland pituitary insufficiency plasma Plate posterior potassium POUCH primary produce protein radioactive iodine rare renal secretion sella serum sexual sinus sodium STAIN stimulation suprarenal symptoms syndrome therapy thyroid gland thyroid hormone thyrotropic tion tissue tubule tumor uptake urinary urine usually vasopressin vitamin