Inflammatory Bowel DiseaseJoseph B. Kirsner World-renowned experts present state-of-the-art information in the clinical understanding of inflammatory bowel disease in the 5th Edition of this best-selling text. Scholarly and pragmatic, this text features authoritative information on the medical and surgical management of IBD with the support of over 390 illustrations. Features many new chapters, including those covering intestinal permeability in health and disease, the intestinal response to acute inflammation and injuries, and current concepts of IBD etiology and pathogenesis. |
From inside the book
Results 1-3 of 80
Page 114
... acid . Recently , a bystander functional ileal injury has been shown to occur , independent of contact , follow- ing acetic acid induction of pancolitis.14 Adaptations of this colitis model have extended to the use of 7 % acetic acid in ...
... acid . Recently , a bystander functional ileal injury has been shown to occur , independent of contact , follow- ing acetic acid induction of pancolitis.14 Adaptations of this colitis model have extended to the use of 7 % acetic acid in ...
Page 115
... acid metabolites in acetic acid - induced colitis are identical to those seen in human ulcerative colitis and probably occur as a consequence of neutrophil infiltration.11 Changes in myeloperoxidase ( MPO ) ac- tivity in colonic tissue ...
... acid metabolites in acetic acid - induced colitis are identical to those seen in human ulcerative colitis and probably occur as a consequence of neutrophil infiltration.11 Changes in myeloperoxidase ( MPO ) ac- tivity in colonic tissue ...
Page 754
... acid secretion could account for the increased gastric acid secretion . Whatever the mechanism , this gastric hypersecretory state is transient , improves with intes- tinal adaptation , and generally is well controlled with H2 ...
... acid secretion could account for the increased gastric acid secretion . Whatever the mechanism , this gastric hypersecretory state is transient , improves with intes- tinal adaptation , and generally is well controlled with H2 ...
Common terms and phrases
acid activity acute addition adhesion agents altered animals antibodies antigen appears associated bacterial barrier cause changes chronic Clin clinical colonic common complex Crohn's disease cytokines defect demonstrated develop effect enhanced enteric epithelial cells epithelium et al evidence experimental expression factor function Gastroenterol Gastroenterology gene genetic growth factor human immune Immunol important incidence increased induced infection inflammation inflammatory bowel disease inhibition initial injury interactions intestinal epithelial cells involved lamina propria levels lymphocytes lymphoid macrophages major mechanisms mediators mice molecules mucosal muscle neutrophils normal observed occurs oral patients permeability Peyer's patches population present produced protective protein rates rats recent receptor region regulation relatives reported response risk role secretion severe showed shown similar small intestine smoking specific stimulation studies suggest surface T-cell Table tion tissue tolerance treatment ulcerative colitis