Inflammatory Bowel DiseaseJoseph B. Kirsner World-renowned experts present state-of-the-art information in the clinical understanding of inflammatory bowel disease in the 5th Edition of this best-selling text. Scholarly and pragmatic, this text features authoritative information on the medical and surgical management of IBD with the support of over 390 illustrations. Features many new chapters, including those covering intestinal permeability in health and disease, the intestinal response to acute inflammation and injuries, and current concepts of IBD etiology and pathogenesis. |
From inside the book
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Page 124
... chronic phases of inflammation are associated with activation of the kallikrein sys- tem130 and with increased cecal levels of IL - 1 , IL - 6 , the anti - inflammatory cytokine IL - 1 receptor antagonist ( IL - 1RA ) , the inhibitory ...
... chronic phases of inflammation are associated with activation of the kallikrein sys- tem130 and with increased cecal levels of IL - 1 , IL - 6 , the anti - inflammatory cytokine IL - 1 receptor antagonist ( IL - 1RA ) , the inhibitory ...
Page 128
... chronic mucosal Colon , 3-4 mo distal > proximal Interleukin - 2 gene Mice 100 % ; species Acute , chronic Colon 6-15 wk deficient dependent Interleukin - 10 gene Mice 100 % ; species deficient dependent mucosal Acute , chronic ...
... chronic mucosal Colon , 3-4 mo distal > proximal Interleukin - 2 gene Mice 100 % ; species Acute , chronic Colon 6-15 wk deficient dependent Interleukin - 10 gene Mice 100 % ; species deficient dependent mucosal Acute , chronic ...
Page 172
... chronic idiopathic inflammatory bowel diseases ( IBD ) driven by ubiquitous luminal products . Susceptibility factors include genetically determined mucosal barrier function and regulation of the mucosal immune response . ( From Schultz ...
... chronic idiopathic inflammatory bowel diseases ( IBD ) driven by ubiquitous luminal products . Susceptibility factors include genetically determined mucosal barrier function and regulation of the mucosal immune response . ( From Schultz ...
Common terms and phrases
acid activity acute addition adhesion agents altered animals antibodies antigen appears associated bacterial barrier cause changes chronic Clin clinical colonic common complex Crohn's disease cytokines defect demonstrated develop effect enhanced enteric epithelial cells epithelium et al evidence experimental expression factor function Gastroenterol Gastroenterology gene genetic growth factor human immune Immunol important incidence increased induced infection inflammation inflammatory bowel disease inhibition initial injury interactions intestinal epithelial cells involved lamina propria levels lymphocytes lymphoid macrophages major mechanisms mediators mice molecules mucosal muscle neutrophils normal observed occurs oral patients permeability Peyer's patches population present produced protective protein rates rats recent receptor region regulation relatives reported response risk role secretion severe showed shown similar small intestine smoking specific stimulation studies suggest surface T-cell Table tion tissue tolerance treatment ulcerative colitis