Inflammatory Bowel DiseaseJoseph B. Kirsner World-renowned experts present state-of-the-art information in the clinical understanding of inflammatory bowel disease in the 5th Edition of this best-selling text. Scholarly and pragmatic, this text features authoritative information on the medical and surgical management of IBD with the support of over 390 illustrations. Features many new chapters, including those covering intestinal permeability in health and disease, the intestinal response to acute inflammation and injuries, and current concepts of IBD etiology and pathogenesis. |
From inside the book
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Page 56
... Endothelial Cell Interactions Leukocyte adhesion to microvascular endothelium involves a complex sequence of interactions between circulating leukocytes and vascular endothelial cells ( Fig . 5-1 ) . The initial event is believed to be ...
... Endothelial Cell Interactions Leukocyte adhesion to microvascular endothelium involves a complex sequence of interactions between circulating leukocytes and vascular endothelial cells ( Fig . 5-1 ) . The initial event is believed to be ...
Page 57
... Endothelial Cell Interactions Constitutive Expression Immune - Epithelial Cell Interactions Ian R. Sanderson , M.D. ... Endothelial Yes Yes L - selectin Rolling GMP - 140 cells PSGL - 1 CD62P Platelets 120 kDa PSL E - selectin ELAM - 1 ...
... Endothelial Cell Interactions Constitutive Expression Immune - Epithelial Cell Interactions Ian R. Sanderson , M.D. ... Endothelial Yes Yes L - selectin Rolling GMP - 140 cells PSGL - 1 CD62P Platelets 120 kDa PSL E - selectin ELAM - 1 ...
Page 58
... endothelium ( despite the shear stress of flowing blood ) by virtue of the strong adhesive interactions between ẞ2 integrins ( CD11 / CD18 ) on these leuko- cytes and ICAM - 1 on endothelial cells ( see Table 5-1 ) . In vivo studies ...
... endothelium ( despite the shear stress of flowing blood ) by virtue of the strong adhesive interactions between ẞ2 integrins ( CD11 / CD18 ) on these leuko- cytes and ICAM - 1 on endothelial cells ( see Table 5-1 ) . In vivo studies ...
Common terms and phrases
acid activity acute addition adhesion agents altered animals antibodies antigen appears associated bacterial barrier cause changes chronic Clin clinical colonic common complex Crohn's disease cytokines defect demonstrated develop effect enhanced enteric epithelial cells epithelium et al evidence experimental expression factor function Gastroenterol Gastroenterology gene genetic growth factor human immune Immunol important incidence increased induced infection inflammation inflammatory bowel disease inhibition initial injury interactions intestinal epithelial cells involved lamina propria levels lymphocytes lymphoid macrophages major mechanisms mediators mice molecules mucosal muscle neutrophils normal observed occurs oral patients permeability Peyer's patches population present produced protective protein rates rats recent receptor region regulation relatives reported response risk role secretion severe showed shown similar small intestine smoking specific stimulation studies suggest surface T-cell Table tion tissue tolerance treatment ulcerative colitis