Inflammatory Bowel DiseaseJoseph B. Kirsner World-renowned experts present state-of-the-art information in the clinical understanding of inflammatory bowel disease in the 5th Edition of this best-selling text. Scholarly and pragmatic, this text features authoritative information on the medical and surgical management of IBD with the support of over 390 illustrations. Features many new chapters, including those covering intestinal permeability in health and disease, the intestinal response to acute inflammation and injuries, and current concepts of IBD etiology and pathogenesis. |
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Page 212
... lamina propria . The entry of naive cells into Peyer's patches requires additional ligand- receptor interactions ( Fig . 14-3 ) . Another ligand recep- tor interaction , between E cadherin on epithelial cells and aẞ7 integrins on ...
... lamina propria . The entry of naive cells into Peyer's patches requires additional ligand- receptor interactions ( Fig . 14-3 ) . Another ligand recep- tor interaction , between E cadherin on epithelial cells and aẞ7 integrins on ...
Page 219
... lamina propria cells isolated from inflamed versus noninflamed mucosa , although there are differences between lamina propria cells versus peripheral blood cells , with a tendency for lamina propria cells to respond less well . Indeed ...
... lamina propria cells isolated from inflamed versus noninflamed mucosa , although there are differences between lamina propria cells versus peripheral blood cells , with a tendency for lamina propria cells to respond less well . Indeed ...
Page 220
... lamina propria cells from control subjects and patients with IBD , 158 and anti - CD3 me- diated redirected lysis is present in normal lamina propria cells.50 Both of these assays reflect cytotoxic T - cell capability in the mucosa ...
... lamina propria cells from control subjects and patients with IBD , 158 and anti - CD3 me- diated redirected lysis is present in normal lamina propria cells.50 Both of these assays reflect cytotoxic T - cell capability in the mucosa ...
Common terms and phrases
acid activity acute addition adhesion agents altered animals antibodies antigen appears associated bacterial barrier cause changes chronic Clin clinical colonic common complex Crohn's disease cytokines defect demonstrated develop effect enhanced enteric epithelial cells epithelium et al evidence experimental expression factor function Gastroenterol Gastroenterology gene genetic growth factor human immune Immunol important incidence increased induced infection inflammation inflammatory bowel disease inhibition initial injury interactions intestinal epithelial cells involved lamina propria levels lymphocytes lymphoid macrophages major mechanisms mediators mice molecules mucosal muscle neutrophils normal observed occurs oral patients permeability Peyer's patches population present produced protective protein rates rats recent receptor region regulation relatives reported response risk role secretion severe showed shown similar small intestine smoking specific stimulation studies suggest surface T-cell Table tion tissue tolerance treatment ulcerative colitis