Inflammatory Bowel DiseaseJoseph B. Kirsner World-renowned experts present state-of-the-art information in the clinical understanding of inflammatory bowel disease in the 5th Edition of this best-selling text. Scholarly and pragmatic, this text features authoritative information on the medical and surgical management of IBD with the support of over 390 illustrations. Features many new chapters, including those covering intestinal permeability in health and disease, the intestinal response to acute inflammation and injuries, and current concepts of IBD etiology and pathogenesis. |
From inside the book
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Page 183
... macrophages with various effector capabili- ties.42 , 43 Many classes of tissue macrophages express different surface antigens and have different func- tional capabilities ( proliferation , phagocytosis , tumor cell killing , etc ...
... macrophages with various effector capabili- ties.42 , 43 Many classes of tissue macrophages express different surface antigens and have different func- tional capabilities ( proliferation , phagocytosis , tumor cell killing , etc ...
Page 227
... macrophage function as a primary factor in the patho- genesis of IBD.273 Also , drugs effective in the treatment of IBD inhibit monocyte / macrophage function . Intestinal Macrophages Macrophages form a significant proportion of the ...
... macrophage function as a primary factor in the patho- genesis of IBD.273 Also , drugs effective in the treatment of IBD inhibit monocyte / macrophage function . Intestinal Macrophages Macrophages form a significant proportion of the ...
Page 228
... macrophages in the normal and inflamed intestinal mucosa . Resident macrophages in the normal , noninflamed mucosa have attenuated function , possibly due to microenvironmental effects . These cells are phagocytic but do not express the ...
... macrophages in the normal and inflamed intestinal mucosa . Resident macrophages in the normal , noninflamed mucosa have attenuated function , possibly due to microenvironmental effects . These cells are phagocytic but do not express the ...
Common terms and phrases
acid activity acute addition adhesion agents altered animals antibodies antigen appears associated bacterial barrier cause changes chronic Clin clinical colonic common complex Crohn's disease cytokines defect demonstrated develop effect enhanced enteric epithelial cells epithelium et al evidence experimental expression factor function Gastroenterol Gastroenterology gene genetic growth factor human immune Immunol important incidence increased induced infection inflammation inflammatory bowel disease inhibition initial injury interactions intestinal epithelial cells involved lamina propria levels lymphocytes lymphoid macrophages major mechanisms mediators mice molecules mucosal muscle neutrophils normal observed occurs oral patients permeability Peyer's patches population present produced protective protein rates rats recent receptor region regulation relatives reported response risk role secretion severe showed shown similar small intestine smoking specific stimulation studies suggest surface T-cell Table tion tissue tolerance treatment ulcerative colitis