Inflammatory Bowel DiseaseJoseph B. Kirsner World-renowned experts present state-of-the-art information in the clinical understanding of inflammatory bowel disease in the 5th Edition of this best-selling text. Scholarly and pragmatic, this text features authoritative information on the medical and surgical management of IBD with the support of over 390 illustrations. Features many new chapters, including those covering intestinal permeability in health and disease, the intestinal response to acute inflammation and injuries, and current concepts of IBD etiology and pathogenesis. |
From inside the book
Results 1-3 of 95
Page 3
... pathogens and noxious agents Increased enterocyte MHC. 1 Eugene B. Chang , M.D. Mucosal inflammation of the small and large bowel is an important physiologic response to enteric patho- gens , serving as a deterrent to systemic entry of ...
... pathogens and noxious agents Increased enterocyte MHC. 1 Eugene B. Chang , M.D. Mucosal inflammation of the small and large bowel is an important physiologic response to enteric patho- gens , serving as a deterrent to systemic entry of ...
Page 4
... pathogen CI Paneth cells cryptdin Cr Cytokines Chemokines CH Immune and inflammatory Recruitment mediators and activation of immune and inflammatory cells IqA Figure 1-1 Role of intestinal epithelial cells in host response to pathogens ...
... pathogen CI Paneth cells cryptdin Cr Cytokines Chemokines CH Immune and inflammatory Recruitment mediators and activation of immune and inflammatory cells IqA Figure 1-1 Role of intestinal epithelial cells in host response to pathogens ...
Page 156
... Pathogens In contrast to the controversy surrounding persis- tent infections causing IBD , there is abundant evidence that common pathogens can precede the onset of IBD , exacerbate symptoms , and reactivate quiescent dis- ease.21 These ...
... Pathogens In contrast to the controversy surrounding persis- tent infections causing IBD , there is abundant evidence that common pathogens can precede the onset of IBD , exacerbate symptoms , and reactivate quiescent dis- ease.21 These ...
Common terms and phrases
acid activity acute addition adhesion agents altered animals antibodies antigen appears associated bacterial barrier cause changes chronic Clin clinical colonic common complex Crohn's disease cytokines defect demonstrated develop effect enhanced enteric epithelial cells epithelium et al evidence experimental expression factor function Gastroenterol Gastroenterology gene genetic growth factor human immune Immunol important incidence increased induced infection inflammation inflammatory bowel disease inhibition initial injury interactions intestinal epithelial cells involved lamina propria levels lymphocytes lymphoid macrophages major mechanisms mediators mice molecules mucosal muscle neutrophils normal observed occurs oral patients permeability Peyer's patches population present produced protective protein rates rats recent receptor region regulation relatives reported response risk role secretion severe showed shown similar small intestine smoking specific stimulation studies suggest surface T-cell Table tion tissue tolerance treatment ulcerative colitis